May 2004
Volume 45, Issue 13
Free
ARVO Annual Meeting Abstract  |   May 2004
ACTIVATION OF p53 PATHWAY FOLLOWING ISCHEMIA/REPERFUSION IN RAT RETINA.
Author Affiliations & Notes
  • K. Patil
    Department of Ophthalmology,
    New York Medical College, Valhalla, NY
  • D. Vazan
    New York Medical College, Valhalla, NY
  • V. Sarup
    Department of Ophthalmology,
    New York Medical College, Valhalla, NY
  • S. Sharma
    Department of Ophthalmology,
    New York Medical College, Valhalla, NY
  • Footnotes
    Commercial Relationships  K. Patil, None; D. Vazan, None; V. Sarup, None; S. Sharma, None.
  • Footnotes
    Support  PHS grant EY 11295
Investigative Ophthalmology & Visual Science May 2004, Vol.45, 3267. doi:
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      K. Patil, D. Vazan, V. Sarup, S. Sharma; ACTIVATION OF p53 PATHWAY FOLLOWING ISCHEMIA/REPERFUSION IN RAT RETINA. . Invest. Ophthalmol. Vis. Sci. 2004;45(13):3267.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Abstract: : Purpose:To determine the role of p53 in apoptosis after ischemia/reperfusion in rat retina. To assess the role of p53 inhibitor (Pifithrin α, Calbiochem, CA) and evaluate changes in Bax, one of the pro–apoptotic target protein of p53 after ischemia/reperfusion in rat retina. Methods:Pifithrin α (0.5mg) and vehicle was injected intraperitoneally 30 minutes before induction of ischemia (75 minutes) followed by reperfusion. Animals were sacrificed at 1 day, 2 day and 3 days after ischemia/reperfusion. Retinas were processed for p53 and Bax proteins analysis by western blot. As a corollary experiment, HeLa cells were subjected to doxorubicin (to induce p53) and Pifithrin α treatment and were analyzed by western blot for p53. Results:The amount of p53 protein increased at all time points after ischemia/reperfusion in the retina. The amount of p53 protein decreased after the treatment with the Pifithrin α at 1, 2 and 3 days after ischemia/reperfusion. Bax protein significantly reduced after 2 and 3 days of ischemia/ reperfusion with Pifithrin α treatment however, no appreciable difference was noticed at 1 day after ischemia/reperfusion. The amount of p53 protein was significantly reduced in doxorubicin treated HeLa cells after treatment with Pifithrin α. Conclusion:P53 protein is upregulated after the ischemia/reperfusion in the rat retina. Pifithrin α treatment reduces the Bax protein (down stream activator of apoptosis) expression in retina after ischemia reperfusion suggesting the role of p53 in retinal apoptosis.

Keywords: apoptosis/cell death • ischemia • retina 
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