Purchase this article with an account.
F Boscia, N Cardascia, L Sborgia, N Recchimurzo, T Micelli Ferrari, C Sborgia; Evaluation Of Corneal Damage By Passive Efflux Of Silicone Oil In Vitrectomized Eyes . Invest. Ophthalmol. Vis. Sci. 2002;43(13):634.
Download citation file:
© ARVO (1962-2015); The Authors (2016-present)
Purpose: A theoretical concern exists regarding the damage caused to corneal endothelium by the oil flowing across the anterior chamber and out of the eye during passive silicone oil drainage after pars plana vitrectomy. We evaluated the damage to corneal endothelium caused by combined cataract and passive silicone oil removal. Methods: This study included 15 consecutive patients (15 eyes; 12 men, 3 women) with a mean age of 56 years ± 11.2 (SD) who had phacoemulsification combined with silicone oil removal through planned posterior capsulorhexis from January 2000 to June 2001 at the Department of Ophthalmology and Otolaryngology, University of Bari, Bari, Italy. A comparable control group of 12 patients (12 eyes; 6 men, 6 women) with a mean age of 59.8 years ± 11.6 (SD) underwent phacoemulsification and posterior capsule IOL implant. All the procedures in the two groups were performed under topical anesthesia (oxybuprocaine hydrochloride 0.4%), by the same surgeon (FB). Pre and postoperative best-corrected visual acuity (BCVA), endothelial cell count and ultrasound pachymetry (AlconR Surgical Pachymeter) were compared between the 2 groups. Results: After a mean follow-up of 5 months (range 2-18 months), mean BCVA improved in both the groups. In the phaco/ROSO group the mean endothelial cell loss was 264.5 ± 451.7 cell/mm2; in the phaco/IOL group the mean loss was 482 ± 531.3 cell/mm2 (p=0.26, un-paired t-test). Mean pachymetric thickness increased significantly postoperatively in the first group from 556.2 ± 32.1 m to 608.0 ± 51.9 m, and in the second group from 553.0 ± 35.7 m to 571.0 ± 36.0 m ( p=0.053, un-paired t-test). Conclusion: No cases of postoperative keratopathy nor corneal edema was encountered in our series. Even if the passive silicone oil efflux causes a significant endothelial cell loss and an increase in corneal thickness, these modifications do not differ from the insult of a standard phacoemulsification with posterior capsule IOL implant.
This PDF is available to Subscribers Only