June 2013
Volume 54, Issue 15
Free
ARVO Annual Meeting Abstract  |   June 2013
Effects of intravitreal ranibizumab on retinal hard exudates in patients from the RIDE & RISE diabetic macular edema trials
Author Affiliations & Notes
  • Michael Ip
    Ophthalmology, Univ of Wisconsin-Madison, Madison, WI
  • Amitha Domalpally
    Ophthalmology, Univ of Wisconsin-Madison, Madison, WI
  • Dafeng Chen
    Genentech, Inc., South San Francisco, CA
  • Jason Ehrlich
    Genentech, Inc., South San Francisco, CA
  • Footnotes
    Commercial Relationships Michael Ip, Eye Technology Ltd. (C), Genentech, Inc. (C), NicOx (C), Notal Vision (C), QLT Phototherapeutics, Inc. (C), Regeneron (C), Sirion (C), Allergan, Inc (F), Regeneron (F); Amitha Domalpally, None; Dafeng Chen, Genentech/Roche (F), Genentech/Roche (C); Jason Ehrlich, Genentech (E), Roche (I)
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2013, Vol.54, 2394. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Michael Ip, Amitha Domalpally, Dafeng Chen, Jason Ehrlich; Effects of intravitreal ranibizumab on retinal hard exudates in patients from the RIDE & RISE diabetic macular edema trials. Invest. Ophthalmol. Vis. Sci. 2013;54(15):2394.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: Hard exudates (HE) represent lipid-rich extravascular deposits in the retina that are frequently seen with diabetic macular edema (DME). The presence and growth of HE, particularly near the fovea, is thought to be associated with an increased risk of visual impairment. We analyzed the effect of anti-VEGF therapy with ranibizumab on HE lesions in participants from the RIDE and RISE Phase III trials.

Methods: A total of 759 patients with DME were randomized 1:1:1 to receive monthly 0.3 mg or 0.5 mg ranibizumab, or sham injections. Fundus photographs were graded by a central reading center. The total area of HE in the central, inner and outer subfields of the study eye was categorized as absent (absent or questionable) and present (definite, obvious, moderate or severe). Non-gradable photographs were excluded as missing. Change in total area of HE from baseline was calculated in mm2.

Results: Data from 739 patients were available for analysis. Through 24 months of treatment the percentages of study eyes in the absent category increased from 20.9% to 36.5% in the sham arm and from 22.1% to 60.7% and 23.6% to 61.2% in the ranibizumab 0.3 and 0.5 mg arms respectively. The resolution of DME was not paralleled by an increase in HE. Decrease in percentage of study eyes in the HE present category was evident after 6 months in the ranibizumab arms. At baseline, there was no correlation between VA and presence of HE in the central subfield in any treatment arm. Post-baseline, there was no consistent correlation between presence of HE in the central subfield and VA change over time.

Conclusions: In this exploratory analysis, ranibizumab appears to result in a reduction in the distribution of HE in patients with DME. However, baseline VA was not associated with the presence of HE, nor was the therapeutic benefit of ranibizumab on visual acuity clearly associated with the presence or absence of HE in the central subfield. Additionally, contrary to prior expectations, the presence and area of HE did not increase as DME resolved (both in the ranibizumab or sham groups).

Keywords: 499 diabetic retinopathy • 505 edema • 688 retina  
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×