June 2013
Volume 54, Issue 15
Free
ARVO Annual Meeting Abstract  |   June 2013
Intravitreal Bevacizumab for Choroidal Neovascularization due to Pathological Myopia: Long-term Outcomes
Author Affiliations & Notes
  • Valentina Sarao
    Department of Ophthalmology, University of Udine, Udine, Italy
  • Daniele Veritti
    Department of Ophthalmology, University of Udine, Udine, Italy
  • Sara Macor
    Department of Ophthalmology, University of Udine, Udine, Italy
  • Paolo Lanzetta
    Department of Ophthalmology, University of Udine, Udine, Italy
  • Footnotes
    Commercial Relationships Valentina Sarao, None; Daniele Veritti, None; Sara Macor, None; Paolo Lanzetta, Alimera (C), Allergan (C), Bayer (C), Novartis (C), Novartis (R), Roche (C), Iridex (P)
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2013, Vol.54, 2796. doi:
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      Valentina Sarao, Daniele Veritti, Sara Macor, Paolo Lanzetta; Intravitreal Bevacizumab for Choroidal Neovascularization due to Pathological Myopia: Long-term Outcomes. Invest. Ophthalmol. Vis. Sci. 2013;54(15):2796.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose
 

To report long-term results from intravitreal bevacizumab (IVB) treatment of choroidal neovascularization (CNV) secondary to pathological myopia (PM).

 
Methods
 

A retrospective review of eyes affected by CNV due to PM and treated with IVB (1 mg/0.04 ml) was conducted. Indication for treatment was an angiographically active lesion with/without intra- or sub-retinal fluid accumulation on spectral domain optical coherence tomography (SD-OCT). Patients received an injection at baseline and were then followed-up for a period of 60 months. Patients were scheduled for follow-up examinations at 1, 3 and 6 months after each treatment. Extra visits were planned upon patient request due to symptoms. Best-corrected visual acuity (BCVA, LogMAR), fluorescein angiography (FA) and SD-OCT were performed at baseline and at follow-up visits. Re-treatment was considered at investigator discretion at every follow-up visit. The primary efficacy outcome was change in BCVA. Secondary outcomes include the reduction in central retinal thickness (CRT) and the number of injections required during the follow-up. The incidence of ocular and non-ocular adverse events was recorded.

 
Results
 

The study included 101 consecutive eyes of 86 patients with CNV secondary to PM. Data from a 24-month follow-up are available for all patients. Thirty-two eyes reached 60 months of follow up. CNVs were located subfoveally in all eyes. Thirty-five percent of patients had received photodynamic therapy before enrollment. Mean BCVA improvement at 24 months was 0.13 (95%CI: 0.05; 0.2) logMAR (p<0.001). Retinal thickness decreased on average by 67 (95%CI: 27; 102) µm after 2 years of follow-up (p<0.01). On average, patients received 4.1 treatments in 24 months. Visual acuity improved on average by 0.05 (95%CI: -0.1; 0.2) logMAR (p>0.05) at 60 months. Mean reduction in CRT was 102 (95%CI: 64;141) µm after 5 years of follow-up (p<0.01). Patients received a mean of 6.7 treatments in 60 months. No significant adverse events were recorded during the follow up.

 
Conclusions
 

Intravitreal VEGF inhibition is an effective therapy for the treatment of myopic CNV. A pro-re-nata regimen with intravitreal bevacizumab has produced functional and morphological improvements in the mid and long-term period.

 
Keywords: 605 myopia • 453 choroid: neovascularization • 748 vascular endothelial growth factor  
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