June 2013
Volume 54, Issue 15
Free
ARVO Annual Meeting Abstract  |   June 2013
Early Retinal Ganglion Cell Layer Thinning Due to Acute NAION and Optic Neuritis
Author Affiliations & Notes
  • Mark Kupersmith
    Neuro-Ophthalmology, Roosevelt Hospital and NYEE, New York, NY
  • Mona Garvin
    Electrical and Computer Engineering and Center, University of Iowa, Iowa City, IA
    Center of Excellence for the Prevention and Treatment of Visual Loss, Department of Veterans Affairs, Rehabilitation Research and Development Division, Iowa City, IA
  • Jui-Kai Wang
    Electrical and Computer Engineering and Center, University of Iowa, Iowa City, IA
  • Randy Kardon
    Center of Excellence for the Prevention and Treatment of Visual Loss, Department of Veterans Affairs, Rehabilitation Research and Development Division, Iowa City, IA
    Ophthalmology, University of Iowa, Iowa City, IA
  • Footnotes
    Commercial Relationships Mark Kupersmith, None; Mona Garvin, Patent application 12/001,066 (P); Jui-Kai Wang, None; Randy Kardon, Novartis (C), Acorda (C), Fight for Sight Inc (S), Department of Veterans Affairs Research Foundation Iowa City, IA (S)
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2013, Vol.54, 3233. doi:
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    • Get Citation

      Mark Kupersmith, Mona Garvin, Jui-Kai Wang, Randy Kardon; Early Retinal Ganglion Cell Layer Thinning Due to Acute NAION and Optic Neuritis. Invest. Ophthalmol. Vis. Sci. 2013;54(15):3233.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose
 

OCT shows retinal ganglion cell layer (GCL) loss correlation with RNFL thinning and vision loss due to glaucoma and multiple sclerosis but it has not been studied with acute optic neuropathy. We hypothesize that GCL loss occurs before RNFL loss detection, particularly with optic nerve head (ONH) swelling. GCL could provide another structure-function interaction to evaluate acute optic nerve injury.

 
Methods
 

We prospectively studied eyes with new onset NAION (36) and optic neuritis [(ON) 28] using SAP and HD OCT volume images centered on the ONH and macula. Using The University of Iowa graph-theoretic approach for layer segmentation, the combined GCL and inner-plexiform-layer (GCL+IPL) thickness was computed within an elliptical annulus centered at the fovea (with a vertical inner and outer radius of 0.5 mm and 2.0 mm, respectively; and horizontal inner and outer radius of 0.6 mm and 2.4 mm, respectively). We report average RNFL and macula GCL+IPL values at presentation and change at 1 month.

 
Results
 

At presentation, the mean for the average GCL+IPL value for affected eyes, 80 µm + 8.1 for NAION and 83 µm + 8.9 for ON, did not differ from unaffected fellow eyes (83 + 6.4 and 82 + 7.0; NS). GCL thinning worsened in both groups over time (Figure: NAION pattern, ON white). At presentation, 5 NAION and 2 ON eyes had GCL+IPL < 5th percentile of unaffected eyes. RNFL thinning <5th percentile of controls (in none at presentation) developed in 4 NAION and 3 ON eyes at 1 month. At 1 month, 29 NAION and 19 ON eyes had GCL+IPL thinning > 3.2 µm (95th percentile for retest unaffected eyes, mean 0.13 + 1.2 µm), with mean thinning 17.2 µm + 12.3 and 9.0 µm + 6.2, respectively for NAION and ON eyes (p=0.001). At 1 month, GCL+IPL thinning correlated with Logmar acuity (r=0.62, p=0.01) and mean deviation (r=0.60, p=0.01) for NAION but not for ON eyes.

 
Conclusions
 

GCL thinning occurs rapidly in NAION and ON, but it is more profound in NAION. RGC loss occurs prior to RFNL thinning, making it a better biomarker of early structural loss.

  
Keywords: 629 optic nerve • 550 imaging/image analysis: clinical • 531 ganglion cells  
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