June 2013
Volume 54, Issue 15
Free
ARVO Annual Meeting Abstract  |   June 2013
Gene Expression Alterations in Mouse Retina with Deficiency of Cone Cyclic Nucleotide-gated Channel Subunits CNGA3 and CNGB3
Author Affiliations & Notes
  • Hongwei Ma
    The Department of Cell Biology, Univ of Oklahoma Health Sci Ctr, Oklahoma City, OK
  • Arjun Thapa
    The Department of Cell Biology, Univ of Oklahoma Health Sci Ctr, Oklahoma City, OK
  • Lynsie Morris
    The Department of Cell Biology, Univ of Oklahoma Health Sci Ctr, Oklahoma City, OK
  • Stylianos Michalakis
    The Center for Integrated Protein Science Munich (CIPSM) and Department of Pharmacy - Center for Drug Research, Ludwig-Maximilians-Universität München, Munich, Germany
  • Martin Biel
    The Center for Integrated Protein Science Munich (CIPSM) and Department of Pharmacy - Center for Drug Research, Ludwig-Maximilians-Universität München, Munich, Germany
  • Mark Frank
    The Arthritis and Clinical Immunology Program, Oklahoma Medical Research Foundation, Oklahoma City, OK
  • Melissa Bebak
    The Arthritis and Clinical Immunology Program, Oklahoma Medical Research Foundation, Oklahoma City, OK
  • Xi-Qin Ding
    The Department of Cell Biology, Univ of Oklahoma Health Sci Ctr, Oklahoma City, OK
  • Footnotes
    Commercial Relationships Hongwei Ma, None; Arjun Thapa, None; Lynsie Morris, None; Stylianos Michalakis, None; Martin Biel, None; Mark Frank, None; Melissa Bebak, None; Xi-Qin Ding, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2013, Vol.54, 4191. doi:
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      Hongwei Ma, Arjun Thapa, Lynsie Morris, Stylianos Michalakis, Martin Biel, Mark Frank, Melissa Bebak, Xi-Qin Ding; Gene Expression Alterations in Mouse Retina with Deficiency of Cone Cyclic Nucleotide-gated Channel Subunits CNGA3 and CNGB3. Invest. Ophthalmol. Vis. Sci. 2013;54(15):4191.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: The cone photoreceptor cyclic nucleotide-gated (CNG) channel is essential for central and color vision and visual acuity. Mutations in the channel subunits CNGA3 and CNGB3 are associated with achromatopsia and cone dystrophy. We investigated the gene expression profiles in mouse retina with CNG channel deficiency using whole genome expression microarrays.

Methods: As cones comprise only 2-3% of the total photoreceptor population in the wild-type mouse retina, the mouse lines with CNG channel deficiency on a cone-dominant background, i.e., Cnga3-/-/Nrl-/- and Cngb3-/-/Nrl-/- mice, were used in our study. Retinal RNAs were prepared from Cnga3-/-/Nrl-/-, Cngb3-/-/Nrl-/- and Nrl-/- mice (at postnatal 30 days) and analyzed by Illumina microarrays. The microarray data were analyzed using a 5% FDR and functionally evaluated using Ingenuity IPA. QRT-PCR and western blotting were performed to confirm the microarray findings.

Results: Comparative data analysis revealed a total of 105 genes altered in Cnga3-/-/Nrl-/- and 92 in Cngb3-/-/Nrl-/- retinas, relative to Nrl-/- retinas at 1.5-fold change, with 27 genes changed in both genotypes. The differentially expressed genes primarily encode proteins associated with cell signaling, cellular function maintenance, and gene expression. Ingenuity Pathways Analysis identified 26 and 9 canonical pathways in Cnga3-/-/Nrl-/- and Cngb3-/-/Nrl-/- retinas, respectively, with 6 pathways shared. The shared pathways include phototransduction, cAMP/PKA-mediated signaling, endothelin signaling, and EIF2/endoplasmic reticulum stress, whereas the IL-1, CREB, and purine metabolism signaling were found to specifically associate with Cnga3 deficiency.

Conclusions: CNG channel deficiency differentially regulates genes that affect cell processes such as phototransduction, cellular survival and gene expression, and such regulations play a crucial role(s) in the retinal adaptation to impaired cone phototransduction. Though lack of Cnga3 and Cngb3 shares many common pathways, deficiency of Cnga3 causes more significant alterations of gene expression. This work provides insight into how cones respond to impaired phototransduction at the gene expression levels.

Keywords: 535 gene microarray • 695 retinal degenerations: cell biology • 569 ion channels  
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