June 2013
Volume 54, Issue 15
Free
ARVO Annual Meeting Abstract  |   June 2013
Abnormal epithelial basement membrane ultrastructure in corneas with haze after PRK in rabbits
Author Affiliations & Notes
  • Andre Torricelli
    Ophthalmology, Cleveland Clinic, Cleveland, OH
    Ophthalmology, FMUSP, Sao Paulo, Brazil
  • Vivek Singh
    Ophthalmology, Cleveland Clinic, Cleveland, OH
  • Vandana Agrawal
    Ophthalmology, Cleveland Clinic, Cleveland, OH
  • Steven Wilson
    Ophthalmology, Cleveland Clinic, Cleveland, OH
  • Footnotes
    Commercial Relationships Andre Torricelli, None; Vivek Singh, None; Vandana Agrawal, None; Steven Wilson, Allergan (C)
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2013, Vol.54, 5226. doi:
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      Andre Torricelli, Vivek Singh, Vandana Agrawal, Steven Wilson; Abnormal epithelial basement membrane ultrastructure in corneas with haze after PRK in rabbits. Invest. Ophthalmol. Vis. Sci. 2013;54(15):5226.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose
 

To assess the ultrastructure of the cornea epithelial basement membrane (EBM) using transmission electron microscopy (TEM) in corneas with haze after -9D PRK and corneas without haze after -4.5D PRK compared to control corneas in rabbits.

 
Methods
 

Two groups of six rabbits each were included in this study. The first group had -4.5 diopter (D) PRK and the second group had -9.0 D PRK. Photorefractive keratectomy (PRK) was performed with epithelial scrape using a VISX Star S4 IR laser. Contralateral eyes of each animal were used as unwound controls. Rabbits were sacrificed at 4 weeks after surgery. Immunohistochemical analysis was performed to detect the myofibroblast marker alpha-smooth muscle actin (SMA). TEM was performed to analyze the ultrastructure of the epithelial basement membrane in PRK and control corneas.

 
Results
 

At 4 weeks after PRK, high numbers of alpha-SMA+ myofibroblasts were observed in the subepithelial stroma of rabbit eyes that had -9.0 D PRK whereas few myofibroblasts were noted in the subepithelial stroma of eyes that had -4.5 D PRK. At one month after -9D PRK, the epithelial basement membrane was irregular and discontinuous and lacking in typical morphology in all four corneas in the group (Figure) compared to the epithelial basement membrane in the four corneas in the -4.5D PRK group or 4 corneas in the control group.

 
Conclusions
 

Transmission electron microscopy at 4 weeks after haze generating -9D PRK in rabbits reveals structural defects in the regenerated epithelial basement membrane that are not observed in corneas that had -4.5D or control corneas. These structural defects likely correlate with decreased basement membrane function that increases penetration of epithelium-derived growth factors such as TGF beta, which both drive development of stromal myofibroblasts from precursor cells and maintain myofibroblast viability. The epithelial basement membrane is a critical regulator of myofibroblast development and persistence.

  
Keywords: 480 cornea: basic science • 484 cornea: stroma and keratocytes • 765 wound healing  
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