June 2013
Volume 54, Issue 15
Free
ARVO Annual Meeting Abstract  |   June 2013
Genetic Screening of TSPAN12, NDP and FZD4 Genes in Indian Patients with Retinopathy of Prematurity
Author Affiliations & Notes
  • Inderjeet Kaur
    Kallam Anji Reddy Molecular Genetics Lab, LV Prasad Eye Institute, Hyderabad, India
  • sonika rathi
    Kallam Anji Reddy Molecular Genetics Lab, LV Prasad Eye Institute, Hyderabad, India
  • Ganeswara Musada
    Kallam Anji Reddy Molecular Genetics Lab, LV Prasad Eye Institute, Hyderabad, India
  • Subhadra Jalali
    Smt.Kannuri Santhamma Centre for Vitreo Retinal Diseases, LV Prasad Eye Institute, Hyderabad, India
  • Ramesh Kekunnaya
    Jasti V Ramanamma Children’s Eye Care Centre, LV Prasad Eye Institute, Hyderabad, India
  • Pramod Gaddam
    Fernandez Hospital, Hyderabad, India
  • Subhabrata Chakrabarti
    Kallam Anji Reddy Molecular Genetics Lab, LV Prasad Eye Institute, Hyderabad, India
  • Footnotes
    Commercial Relationships Inderjeet Kaur, None; sonika rathi, None; Ganeswara Musada, None; Subhadra Jalali, None; Ramesh Kekunnaya, None; Pramod Gaddam, None; Subhabrata Chakrabarti, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2013, Vol.54, 6204. doi:
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      Inderjeet Kaur, sonika rathi, Ganeswara Musada, Subhadra Jalali, Ramesh Kekunnaya, Pramod Gaddam, Subhabrata Chakrabarti; Genetic Screening of TSPAN12, NDP and FZD4 Genes in Indian Patients with Retinopathy of Prematurity. Invest. Ophthalmol. Vis. Sci. 2013;54(15):6204.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: Retinopathy of prematurity (ROP) is a vaso-proliferative eye disease in prematurely born infants. The clinical manifestation of this condition exhibits similarites with Familial Exudative Vitreo-retinopathy (FEVR). Genes involved in the Wnt signalling pathway (TSPAN12, NDP, FZD4) have been implicated in retinal neovascularisation among FEVR patients. Thus, a similar molecular mechanism could be envisaged underlying ROP based on its phenotypic similarities with FEVR. In the present study, we aimed to assess the role of TSPAN12, NDP, FZD4 genes in ROP.

Methods: The TSPAN12, NDP and FZD4 genes were screened in ROP patients (n=200) and controls consisting of mature (n=127) and premature babies devoid of ROP (n=147). Screening was accomplished by resequencing using specific sets of primers to amplify the entire coding and untranslated regions (UTR) in these genes using the BigDye chemistry. The observed variants were further validated by restriction digestion and characterized for mutations or polymorphisms.

Results: The TSPAN12 gene screening led to the identification of a heterozygous mutation in a patient with threshold ROP (L119V), a novel SNP (c.*334A>T) and three reported SNPs (rs41623, rs41622, rs189221112) in the study cohort. There were no significant differences in the allele or genotype frequencies of these SNPs between the ROP patients and controls. Two novel variations viz., IVS1+16A>G in the intron 1 and c.1503T>C in 3’UTR region and a previosuly reported change in the 5’UTR region (c.171_184del14bp) were observed in the NDP gene in 3 ROP patients. Extended screening of the FZD4 also revealed two heterozygous variations I192I and I360V and three reported SNPs (rs61735303, rs201168680, rs61749246) in the study cohort. However, a formal genotype-phenotype corelation could not be established due to the low frequencies of the variant alleles in these genes.

Conclusions: These results suggest a minor involvement of TSPAN12, NDP and FZD4 genes with ROP. Unlike FEVR, ROP may be genetically more heterogeneous with multiple alleles with varying magnitudes of effect.

Keywords: 537 gene screening • 706 retinopathy of prematurity • 604 mutations  
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