October 1990
Volume 31, Issue 10
Free
Articles  |   October 1990
Cell-mediated immunity against human retinal extract, S-antigen, and interphotoreceptor retinoid binding protein in onchocercal chorioretinopathy.
Author Affiliations
  • A Van der Lelij
    Department of Ophthalmology, Free University, Utrecht, The Netherlands.
  • A Rothova
    Department of Ophthalmology, Free University, Utrecht, The Netherlands.
  • J S Stilma
    Department of Ophthalmology, Free University, Utrecht, The Netherlands.
  • R Hoekzema
    Department of Ophthalmology, Free University, Utrecht, The Netherlands.
  • A Kijlstra
    Department of Ophthalmology, Free University, Utrecht, The Netherlands.
Investigative Ophthalmology & Visual Science October 1990, Vol.31, 2031-2036. doi:
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      A Van der Lelij, A Rothova, J S Stilma, R Hoekzema, A Kijlstra; Cell-mediated immunity against human retinal extract, S-antigen, and interphotoreceptor retinoid binding protein in onchocercal chorioretinopathy.. Invest. Ophthalmol. Vis. Sci. 1990;31(10):2031-2036.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Autoimmune mechanisms are thought to be involved in the pathogenesis of onchocercal chorioretinopathy. Cell-mediated immune responses to human retinal S-antigen, interphotoreceptor retinoid binding protein (IRBP), and crude retinal extract were investigated in patients with onchocerciasis from Sierra Leone, West Africa using a two-step migration-inhibition factor assay. Patients were subdivided into three groups: (1) without ocular involvement (n = 10), (2) with ocular onchocerciasis limited to the anterior segment (n = 19), and (3) with onchocercal chorioretinopathy (n = 21). A group of endemic controls (n = 25) from Sierra Leone were also studied. The cellular immune response to concanavalin A (Con A) was measured to assess the general capacity of lymphocytes to respond to a mitogen. Four of 50 (8%) patients with onchocerciasis and four of 25 (16%) endemic controls reacted with at least one retinal antigen. From the patients with onchocercal chorioretinopathy two of 21 (10%) showed a positive cellular response. The general mitogen response tested with Con A was positive in all these individuals. A role for an antiretinal autoimmune mechanism in the pathogenesis of onchocercal chorioretinopathy, as studied with human S-antigen, IRBP, or crude retinal extract, could not be shown because the cellular response to these antigens did not differ in patients with or without onchocercal chorioretinopathy or in endemic controls.

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