October 1998
Volume 39, Issue 11
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Articles  |   October 1998
Vitreous treatment of retinal pigment epithelial cells results in decreased expression of FGF-2.
Author Affiliations
  • D M Hunt
    Department of Microbiology and Immunology, University of South Carolina School of Medicine, Columbia 29208, USA.
  • W H Chen
    Department of Microbiology and Immunology, University of South Carolina School of Medicine, Columbia 29208, USA.
  • R C Hunt
    Department of Microbiology and Immunology, University of South Carolina School of Medicine, Columbia 29208, USA.
Investigative Ophthalmology & Visual Science October 1998, Vol.39, 2111-2120. doi:
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      D M Hunt, W H Chen, R C Hunt; Vitreous treatment of retinal pigment epithelial cells results in decreased expression of FGF-2.. Invest. Ophthalmol. Vis. Sci. 1998;39(11):2111-2120.

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Abstract

PURPOSE: Changes in gene expression were investigated after treatment of cultured human retinal pigment epithelial (RPE) cells with vitreous. This may have implications for proliferative diseases such as proliferative vitreoretinopathy. METHODS: Cells were cultured in the presence or absence of human vitreous, and gene expression was examined using the differential display polymerase chain reaction technique. Differentially expressed RNAs were cloned, screened for differential expression, and sequenced. The expression of one of these RNAs (that for fibroblast growth factor [FGF]-2/basic FGF) was examined by in situ hybridization and ribonuclease protection assays. The level of FGF-2 protein was examined by immunoblot analysis. The effects of adding FGF-2 to cells cultured in the presence of vitreous were examined. RESULTS: Treatment of low passage human RPE cells with 25% vitreous resulted in the epithelial-to-fibroblast-like morphologic changes reported by others and in the decreased expression of FGF-2 mRNA and FGF-2 protein. Addition of FGF-2 to cultures at the same time as addition of vitreous prevented some of the effects of vitreous on these cells. CONCLUSIONS: Vitreous treatment of RPE cells in culture results in decreased expression of FGF-2 mRNA and protein. Because supplementation of FGF-2 prevents some of the vitreous-mediated effects, this may indicate that modulation of FGF-2 levels by the vitreous may play an important role in the phenotypic changes seen in RPE cells exposed to vitreous.

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