June 1989
Volume 30, Issue 6
Free
Articles  |   June 1989
Dark adaptation in hyperprolactinemic women.
Author Affiliations
  • A B Fulton
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
  • V Greenwood
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
  • R M Hansen
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
  • M G McGill
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
  • R M Russell
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
  • P A Deuster
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
  • M C Kappelman
    Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts.
Investigative Ophthalmology & Visual Science June 1989, Vol.30, 1177-1180. doi:
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    • Get Citation

      A B Fulton, V Greenwood, R M Hansen, M G McGill, R M Russell, P A Deuster, M C Kappelman; Dark adaptation in hyperprolactinemic women.. Invest. Ophthalmol. Vis. Sci. 1989;30(6):1177-1180.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

In a study to test the hypothesis that hyperprolactinemia is caused by subclinical zinc deficiency, dark-adaptation curves of seven women with primary hyperprolactinemia and seven normal women were measured to assess tissue zinc status. Although plasma zinc levels, final dark-adapted thresholds, and the time courses of rod dark-adaptation did not differ significantly between patients and normal subjects, the median cone plateau of the hyperprolactinemic patients was significantly higher (0.66 log units) than that of normal subjects. It appears unlikely that derangements of vitamin A metabolism, for which zinc is a cofactor, explains this unanticipated and subtle abnormality in dark adaptation of the hyperprolactinemic women.

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