April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
Enhanced Neurogenic Potential of Müller Cells in the Absence of Ephrin-A2/A3
Author Affiliations & Notes
  • Ruilin Zhu
    Ophthalmology, Peking University First Hospital, Key Laboratory of Vision Loss and Restoration, Ministry of Education, Beijing, China
    Schepens Eye Research Institute, Massachusetts Eye and Ear, Harvard Medical School, Boston, MA
  • Kin-Sang Cho
    Schepens Eye Research Institute, Massachusetts Eye and Ear, Harvard Medical School, Boston, MA
  • Yuan Fang
    Eye and ENT hospital, Fudan University, Shanghai, China
  • Liu Yang
    Ophthalmology, Peking University First Hospital, Key Laboratory of Vision Loss and Restoration, Ministry of Education, Beijing, China
  • Dongfeng Feng Chen
    Schepens Eye Research Institute, Massachusetts Eye and Ear, Harvard Medical School, Boston, MA
    Boston VA Healthcare System, Boson, MA
  • Footnotes
    Commercial Relationships Ruilin Zhu, None; Kin-Sang Cho, None; Yuan Fang, None; Liu Yang, None; Dongfeng Chen, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science April 2014, Vol.55, 1388. doi:
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      Ruilin Zhu, Kin-Sang Cho, Yuan Fang, Liu Yang, Dongfeng Feng Chen; Enhanced Neurogenic Potential of Müller Cells in the Absence of Ephrin-A2/A3. Invest. Ophthalmol. Vis. Sci. 2014;55(13):1388.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: We showed previously that ephrin-A2 and -A3 are negative regulators for the growth of neural progenitor cells in the brain and ciliary epithelium derived retinal stem cells.We hypothesize that absence of ephrin-A2 and -A3 also increases Müller cell proliferation and neurogenic potential in the adult.

Methods: Expression of ephrin-A2 and -A3 and their receptor EphA4 in the retina and Müller cells was assessed by immunostaining and real-time PCR.To label proliferating cells,purified Müller cells of both wild-type and A2-/-A3-/- mice were treated with 0.5μM BrdU for 24 hours in culture.Percentage of BrdU+ cells was then recorded,and expression of retinal progenitor markers was evaluated with real-time PCR.In another series of experiments,purified Müller cells were induced to differentiate in the defined medium for 14 days and stained with primary antibodies against a photoreceptor marker recoverin or retinal ganglion cell marker β-III-tublin for evaluation of their potential of trans-differentiation into retinal neurons.

Results: Expression of ephrin-A2/A3 and their receptor EphA4 was detected in both the retinas and purified Müller cell cultures.Using double-immunolabeling of EphA4 and CRALBP,a marker of Müller cells,in retinal sections we further demonstrate Müller cell expression of EphA4. Moreover,results of real-time PCR confirmed that ephrin-A3 and EphA4 expression is particularly enriched in Müller cells.Expression of neural progenitor cell markers Pax6,Chx10,Ngn2,Sox2 were significantly increased in Müller cells derived from A2-/-A3-/- mice as compared to those of wild-type mice.The percentage of proliferating Müller cells was significantly higher in cultures derived from A2-/-A3-/- mice than that from wild-type mice.Induction of neuron trans-differentiation also induced significantly higher percentage of recoverin+ and β-III-tublin+ cells in Müller cell cultures derived from A2-/-A3-/- mice.These data suggest enhanced neurogenic potential of Müller cells in the absence of ephrin-A2 and-A3.

Conclusions: Our results indicate that the adult mouse retina expresses negative regulators for retinal stem cell growth,ephrin-A2/A3,and their receptor EphA4 on Müller cells.The Müller cells derived from A2-/-A3-/- mice show a higher proliferation and neurogenic potentials in culture than those from wild-type mice.Thus,ephrin-A2 and -A3 contribute negatively to the regenerative behavior of Müller cells in adult mice.

Keywords: 648 photoreceptors • 687 regeneration • 699 retinal glia  
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