April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
Anti-neovascular Effect of Chondrocyte-derived Extracellular Matrix on Corneal Alkaline Burns in Rabbits
Author Affiliations & Notes
  • Hye Sook Lee
    Ocular Neovascular Disease Research Center, Inje University Busan Paik Hospital, Busan, Republic of Korea
  • Ji Hyun Lee
    Ocular Neovascular Disease Research Center, Inje University Busan Paik Hospital, Busan, Republic of Korea
  • Chae Eun Kim
    Ocular Neovascular Disease Research Center, Inje University Busan Paik Hospital, Busan, Republic of Korea
  • JaeWook Yang
    Ocular Neovascular Disease Research Center, Inje University Busan Paik Hospital, Busan, Republic of Korea
    Department of Ophthalmology, Inje University College of Medicine, Busan, Republic of Korea
  • Footnotes
    Commercial Relationships Hye Sook Lee, None; Ji Hyun Lee, None; Chae Eun Kim, None; JaeWook Yang, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science April 2014, Vol.55, 3225. doi:
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      Hye Sook Lee, Ji Hyun Lee, Chae Eun Kim, JaeWook Yang; Anti-neovascular Effect of Chondrocyte-derived Extracellular Matrix on Corneal Alkaline Burns in Rabbits. Invest. Ophthalmol. Vis. Sci. 2014;55(13):3225.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose
 

To investigate the effect of chondrocyte-derived extracellular matrix (CDECM) on experimental corneal alkaline burns in rabbits.

 
Methods
 

Corneal neovascularization (NV) was induced by 1 N NaOH to the right central corneas of rabbits. Ten days later, CDECM and human amniotic membrane (HAM) were transplanted onto the corneal surface to completely cover the resected area and then sutured. On the 10th day after transplantation, the structural changes of the cornea were histologically analyzed. We examined the effects of CDECM on clinical NV features and on the expression of corneal NV markers. This study was conducted in accordance with the Guidelines for Animal Experiments approved by Inje University College of Medicine (No.; 2012-028) and the ARVO Statement for the Use of Animals in Ophthalmic and Vision Research.

 
Results
 

The alkaline burn produced significant NV and increased corneal thickness. On day 10 after transplantation, the thickness, NV and opacity of the cornea were markedly decreased in the CDECM group (p<0.001). However, the HAM transplantation group did not exhibit improvements in these clinical parameters, and there were no significant differences relative to the burn group. In addition, the CDECM improved the healing of the cornea following alkaline burn, disrupting the corneal epithelial proliferation and reducing the fibrotic changes of the stroma. The hallmarks of NV were significantly induced in the subepithelium by the alkaline burn, and these levels were also suppressed by CDECM. The CDECM suppressed corneal NV by inhibiting nuclear factor-kappa B (NF-κB) activation through blocking the PKC and Akt signaling pathway.

 
Conclusions
 

CDECM transplantation was markedly effective in healing alkali-burned corneas by modulating the translocation of NF-kB to the nucleus, making it a promising material for the noninvasive treatment of ocular surface disease.

 
 
Figure 1. Effects of CDECM on clinical outcomes in rabbit cornea with alkali burns.
 
Figure 1. Effects of CDECM on clinical outcomes in rabbit cornea with alkali burns.
 
 
Figure 2. Role of CDECM in the regulation of alkaline burn-induced corneal NV.
 
Figure 2. Role of CDECM in the regulation of alkaline burn-induced corneal NV.
 
Keywords: 609 neovascularization • 519 extracellular matrix • 765 wound healing  
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