April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
Effects of Anti-PDGFRβ Therapy Alone or in Combination with VEGF Trap on Corneal Neovascularization (NV) and Pericyte Recruitment in Mice
Author Affiliations & Notes
  • Yang Liu
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • Zida Li
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • Daniel Reef
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • Ming Yuan
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • Jingtai Cao
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • George D Yancopoulos
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • Stanley J Wiegand
    Regeneron Pharmaceuticals Inc, Tarrytown, NY
  • Footnotes
    Commercial Relationships Yang Liu, Regeneron Pharmaceuticals (E); Zida Li, Regeneron Pharmaceuticals (E); Daniel Reef, Regeneron Pharmaceuticals (E); Ming Yuan, Regeneron Pharmaceuticals (E); Jingtai Cao, Regeneron Pharmaceuticals (E); George Yancopoulos, Regeneron Pharmaceuticals (E); Stanley Wiegand, Regeneron Pharmaceuticals (E)
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science April 2014, Vol.55, 3239. doi:
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      Yang Liu, Zida Li, Daniel Reef, Ming Yuan, Jingtai Cao, George D Yancopoulos, Stanley J Wiegand; Effects of Anti-PDGFRβ Therapy Alone or in Combination with VEGF Trap on Corneal Neovascularization (NV) and Pericyte Recruitment in Mice. Invest. Ophthalmol. Vis. Sci. 2014;55(13):3239.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: To evaluate the effects of anti-PDGFRβ antibody alone or in combination with VEGF Trap on pericyte investment of corneal NV following injury.

Methods: Corneal NV was induced in adult male mice by corneal suture placement or chemical injury. In prevention studies, anti-PDGFRβ antibody (25 mg/kg) was administered subcutaneously (sc) one day before suture placement and then every other day following corneal injury. Control mice received no treatment or an equimolar amount of human Fc (10 mg/kg) following the same schedule. On day 9 after injury, the vasculature was labeled by intravenous injection of fluorescein-conjugated lectin (lycopersicon esculentum) and pericytes were labeled by NG2 immunostaining. Corneal NV and pericyte coverage were evaluated in corneal flat-mounts. Treatment studies evaluated the effects of anti-PDGFRβ or VEGF Trap monotherapy, as well as the effects of combined treatment of anti-PDGFRβ (25 mg/kg, sc, every other day) and VEGF Trap at either efficacious (6.25 mg/kg, sc, every 6 days) or suboptimal doses (0.5 mg/kg or 1.5 mg/kg, sc, every other day). In these studies, treatment was delayed until corneal NV was established (2-3 weeks after injury). Effects of anti-VEGF and anti-PDGFRβ monotherapy, or combined treatment on regression of corneal NV and pericyte coverage were evaluated approximately 2 weeks following the initiation of dosing in corneal flatmounts using the methods described above.

Results: Anti-PDGFRβ monotherapy (25 mg/kg, sc) significantly inhibited pericyte recruitment to newly developing corneal neovessels and also stripped pericytes from established corneal NV, but did not alter pericyte investment of normal corneal limbal vessels in uninjured eyes. Combined treatment with anti-PDGFRβ augmented the anti-angiogenic effects of VEGF inhibition by VEGF Trap, only when low, suboptimal dose (0.5 mg/kg) of VEGF Trap were administered.

Conclusions: Anti-PDGFRβ monotherapy can significantly inhibit pericyte recruitment to developing corneal neovessels and also deplete pericytes from established corneal NV. Combined administration of anti-PDGFRβ and low, suboptimal dose of VEGF Trap produced a significantly greater anti-angiogenic effect than VEGF Trap alone, administered at the same low dose.

Keywords: 543 growth factors/growth factor receptors • 609 neovascularization  
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