April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
Scotopic and Photopic ERG Responses in Pediatric Patients with Usher Syndrome
Author Affiliations & Notes
  • Jena Tavormina
    Ophthalmology, Boston Children's Hospital, Boston, MA
  • Ronald M Hansen
    Ophthalmology, Boston Children's Hospital, Boston, MA
    Harvard Medical School, Boston, MA
  • Anne Moskowitz
    Ophthalmology, Boston Children's Hospital, Boston, MA
    Harvard Medical School, Boston, MA
  • Heidi L Rehm
    Harvard Medical School, Boston, MA
    Brigham and Women's Hospital, Boston, MA
  • Margaret Kenna
    Harvard Medical School, Boston, MA
    Otolaryngology, Boston Children's Hospital, Boston, MA
  • Anne Fulton
    Ophthalmology, Boston Children's Hospital, Boston, MA
    Harvard Medical School, Boston, MA
  • Footnotes
    Commercial Relationships Jena Tavormina, None; Ronald Hansen, None; Anne Moskowitz, None; Heidi Rehm, None; Margaret Kenna, None; Anne Fulton, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science April 2014, Vol.55, 332. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Jena Tavormina, Ronald M Hansen, Anne Moskowitz, Heidi L Rehm, Margaret Kenna, Anne Fulton; Scotopic and Photopic ERG Responses in Pediatric Patients with Usher Syndrome. Invest. Ophthalmol. Vis. Sci. 2014;55(13):332.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: To evaluate scotopic and photopic ERG responses in pediatric patients with a genetic diagnosis of Usher Syndrome, a recessively inherited ciliopathy characterized by hearing loss and retinal degeneration affecting both rods and cones.

Methods: Twenty-two patients (age 2 months to 23 years) with USH2A (n=13) or MYO7A (n=9) disease were studied. ERG responses to a range of full-field scotopic and photopic stimuli (including the ISCEV standard conditions) were recorded and compared to responses in healthy controls (n=72). A model of the activation of phototransduction was used to estimate rod photoreceptor sensitivity (SROD) and saturated amplitude (RROD). Post-receptor b-wave sensitivity was characterized by the stimulus that produced a half maximum response (log σ) and saturated b-wave amplitude (VMAX). Dark adapted thresholds were estimated using a two-alternative, forced choice method in all patients.

Results: Responses to 30 Hz flickering stimuli were detected in all USH2A patients (median 99, range 7 to 169 μV) and all MYO7A patients (median 10, range 1 to 18 μV). Photopic b-wave amplitude was within the normal range in nine of the 13 USH2A patients and none of the nine MYO7A patients. In the seven MYO7A patients whose photopic b-wave was detectable, the responses were less than 20% of the normal mean. Scotopic b-waves were detectable in all USH2A patients but only six MYO7A patients; only four USH2A patients had normal scotopic b-wave amplitudes. Responses were sufficient for estimation of both photoreceptor and post-receptor response parameters in nine USH2A and only two MYO7A patients. For these 11 patients, deficits in scotopic post-receptor b-wave sensitivity were greater than deficits in rod photoreceptor sensitivity. Median dark adapted threshold in the 22 patients was elevated 0.9 (range 0.13-2.77) log units compared to normal and did not differ between USH2A and MYO7A patients. Threshold elevation was outside the 99% prediction interval for normal for 14 of the 22 patients, whereas ERG log σ values were outside the 99% prediction interval for normal for 21 of 22.

Conclusions: The greater deficit in post-receptor than photoreceptor sensitivity is consistent with a ciliopathy. In this sample, ERG log σ was more often abnormal than the dark adapted threshold. Therefore, the ERG may be a more sensitive detector of retinal dysfunction than the dark adapted threshold in children at risk for Usher Syndrome.

Keywords: 509 electroretinography: clinical • 696 retinal degenerations: hereditary • 539 genetics  
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×