April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
The effect of long-term antiglaucomatous drug administration on central corneal thickness
Author Affiliations & Notes
  • Wolfgang A Schrems
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Laura-Maria Schrems-Hoesl
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Christian Y Mardin
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Folkert Horn
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Anselm G M Junemann
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Friedrich E Kruse
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Joachim M Braun
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Robert Laemmer
    Department of Ophthalmology, University of Erlangen-Nuremberg, Erlangen, Germany
  • Footnotes
    Commercial Relationships Wolfgang Schrems, None; Laura-Maria Schrems-Hoesl, None; Christian Mardin, None; Folkert Horn, None; Anselm Junemann, None; Friedrich Kruse, None; Joachim Braun, None; Robert Laemmer, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science April 2014, Vol.55, 4235. doi:
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      Wolfgang A Schrems, Laura-Maria Schrems-Hoesl, Christian Y Mardin, Folkert Horn, Anselm G M Junemann, Friedrich E Kruse, Joachim M Braun, Robert Laemmer; The effect of long-term antiglaucomatous drug administration on central corneal thickness. Invest. Ophthalmol. Vis. Sci. 2014;55(13):4235.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: To evaluate rates of changes per year of central corneal thickness (CCT) after antiglaucomatous drug administration with beta blockers (BB), prostaglandins (PG), and carbonic anhydrase inhibitors (CAI) monotherapy and combined topical antiglaucomatous therapy, in a cohort of patients with ocular hypertension, glaucoma suspects and patients with perimetric glaucoma as compared to normal controls.

Methods: The study included 130 eyes of 66 normal patients, 121 eyes of 65 patients with ocular hypertension, 105 eyes of 63 patients with suspected glaucoma and 49 eyes of 25 patients with perimetric glaucoma (ClinicalTrials.gov number, NTC00494923). All patients underwent standard automated perimetry, 24-hour intraocular pressure profile, optic disc photography and optical coherence pachymetry (OCP, Heidelberg Engineering, Lübeck, Germany). The cohort was divided into 8 groups based on topical antiglaucomatous medication. Linear regression analysis was conducted to analyze the relationship between CCT and exposure to antiglaucomatous medication over time.

Results: There was a statistically significant decrease in CCT for eyes treated with PG, and BB+PG+CAI (-2,1 and -4.9µm/year). CCT raised significantly for eyes treated with CAI (+6.5 µm/year). For both untreated eyes and those treated with BB, BB+CAI, BB+PG, PG+CAI no statistically significant rate of change in CCT was observed.

Conclusions: We recommend regular CCT measurements before and during therapy with PG and PG+BB+CAI and CAI. Follow-up IOP measurements may be underestimated for eyes treated with PG, PG+BB+CAI and overestimated for those treated with CAI if CCT is not measured on a regular basis.

Keywords: 550 imaging/image analysis: clinical • 503 drug toxicity/drug effects • 479 cornea: clinical science  
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