April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
Association Between Genotype and Choroidal Thickness in Reticular Macular Disease (RMD)
Author Affiliations & Notes
  • Nicole Marie Pumariega
    Ophthalmology, New York University School of Medicine, New York, NY
  • Rachel M. Cymerman
    Ophthalmology, New York University School of Medicine, New York, NY
  • Adam Sperber
    Ophthalmology, New York University School of Medicine, New York, NY
  • K Bailey Freund
    Vitreous Retina Macula Consultants of New York, New York, NY
  • Theodore Smith
    Ophthalmology, New York University School of Medicine, New York, NY
  • Footnotes
    Commercial Relationships Nicole Pumariega, None; Rachel Cymerman, None; Adam Sperber, None; K Bailey Freund, None; Theodore Smith, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science April 2014, Vol.55, 5246. doi:
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      Nicole Marie Pumariega, Rachel M. Cymerman, Adam Sperber, K Bailey Freund, Theodore Smith; Association Between Genotype and Choroidal Thickness in Reticular Macular Disease (RMD). Invest. Ophthalmol. Vis. Sci. 2014;55(13):5246.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose
 

RMD has been associated with choroidal thinning and significant progression to advanced AMD. We looked for association between genetics and choroidal thickness (CTh) in RMD, aka subretinal drusenoid deposits (SDD), using spectral domain optical coherence tomography (SD-OCT).

 
Methods
 

We retrospectively identified 29 patients (55 eyes) from a single institution who presented with a reticular pattern in scanning laser ophthalmoscope imaging and SDD in SD-OCT imaging in both eyes. No advanced AMD or large soft drusen were present. Genotyping at the CFH and ARMS2 loci was determined. A total of 55 CTh measurements were taken for each study eye (5 scans/eye,11 fixed points,~550um apart/scan). For most eyes, the five scans included two superior to the fovea, one foveal, and two inferior. We divided patients into 3 groups based on CFH genotype [wild type (T/T), heterozygous (T/C), and homozygous for the risk allele (C/C)] and 3 analogous groups based on ARMS2 genotype. We calculated mean CTh in the superior, foveal, and inferior macula for each group and performed a one-way ANOVA and a paired T test to determine significance.

 
Results
 

The mean age was 84±7 with 83% (24/29) females. At the ARMS2 locus, 7 subjects were wild type, 16 were heterozygous, and 6 were homozygous risk. The corresponding CFH groups numbered, 5, 16 and 8. CTh means for all 55 eyes were 164±59, 160±59, and 157±55 um for the superior, foveal, and inferior macula, respectively. Statistical significance was found when comparing the three CTh means within the ARMS2 genotypes, (P= 0.032; 0.012; 0.006). There also was significance when comparing the CTh means between the ARMS2 wild type (134±39, 124±40, 125±41) versus the homozygous risk (194±59, 189±56, 192±62), (P= 0.034; 0.024; 0.026). There was no statistical significance found between the CTh means within the CFH genotypes.

 
Conclusions
 

ARMS2 genotype, but not CFH genotype, appears to be correlated with CTh in the high risk AMD phenotype of RMD, with increasing load of the risk allele associated with increasing CTh at all locations. To our knowledge, this is the first reported association of an AMD risk allele with choroidal structure. Further study, especially of the relationship with RMD, is warranted.

 
 
Table 1. Choroidal Thickness Measurements
 
Table 1. Choroidal Thickness Measurements
 
Keywords: 412 age-related macular degeneration • 452 choroid • 550 imaging/image analysis: clinical  
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