April 2014
Volume 55, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2014
Optimizing fluence settings and riboflavin composition for collagen cross-linking (CXL) in the antimicrobial efficiency against Pseudomonas aeruginosa and Staphylococcus aureus.
Author Affiliations & Notes
  • Olivier Richoz
    Ophthalmology, Geneva University Hospital, Geneva, Switzerland
  • Florence Hoogewoud
    Ophthalmology, Geneva University Hospital, Geneva, Switzerland
  • David Tabibian
    Ophthalmology, Geneva University Hospital, Geneva, Switzerland
  • Arthur Hammer
    Ophthalmology, Geneva University Hospital, Geneva, Switzerland
  • Farhad Hafezi
    Ophthalmology, Geneva University Hospital, Geneva, Switzerland
    Ophthalmology, Southern California, Doheny Eye Institute, Keck School of Medicine, Los Angeles, CA
  • Footnotes
    Commercial Relationships Olivier Richoz, Emagine AG (I), Emagine AG (P), Emagine AG (S); Florence Hoogewoud, None; David Tabibian, None; Arthur Hammer, None; Farhad Hafezi, Emagine AG (I), Emagine AG (P), Emagine AG (S)
  • Footnotes
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Investigative Ophthalmology & Visual Science April 2014, Vol.55, 5791. doi:
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      Olivier Richoz, Florence Hoogewoud, David Tabibian, Arthur Hammer, Farhad Hafezi; Optimizing fluence settings and riboflavin composition for collagen cross-linking (CXL) in the antimicrobial efficiency against Pseudomonas aeruginosa and Staphylococcus aureus.. Invest. Ophthalmol. Vis. Sci. 2014;55(13):5791.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: When treating bacterial keratitis, determination of the correct pathogen is often clinically challenging. The benefits of using CXL to treat corneal infections is that the treatment is not pathogen-specific. In an attempt to optimize the treatment parameters, we analyzed the effect of high fluence CXL on the bacterial killing rate in an in vitro model using Pseudomonas aeruginosa.

Methods: The killing rate of a known concentration of bacterias (Pseudomonas aeruginosa and Staphylococcus aureus) was analyzed for the following conditions: 1) preservative-free riboflavin, with UV-A irradiation @ 18 mW/cm2 for 5 minutes 2) preservative-free riboflavin, with UV-A irradiation @ 36 mW/cm2 for 2.5 minutes 3) riboflavin with preservatives, with UV-A irradiation @ 18 mW/cm2 for 5 minutes 4) riboflavin with preservatives, with UV-A irradiation @ 36 mW/cm2 for 2.5 minutes 5) riboflavin only, no UVA 6) riboflavin with preservatives, no UVA. We used 0.1% riboflavin in all experiments.

Results: The groups with preservative-free riboflavin showed a killing rate of 2 logs with 18 mW/cm2 and one log with 36 mW/cm2. The groups with riboflavine with preservatives showed a killing rate of 2 logs (98 %) with both fluences.

Conclusions: The P. aeruginosa and S. aureus killing rate is fluence-dependent when using conventional riboflavin and fluence-independent when preservatives are added to the riboflavin solution. These findings will allow the generation of optimized riboflavin solutions for the treatment of bacterial keratitis.

Keywords: 573 keratitis  
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