June 2015
Volume 56, Issue 7
Free
ARVO Annual Meeting Abstract  |   June 2015
In Vivo Confocal Microscopy in Histologically Confirmed Small Fiber Neuropathy
Author Affiliations & Notes
  • Franziska Bucher
    Department of Ophthalmology, University Hospital Cologne, Cologne, Germany
  • Christian Schneider
    Department of Neurology, University Hospital Cologne, Cologne, Germany
  • Tobias Blau
    Department of Pathology, University Hospital Cologne, Cologne, Germany
  • Claus Cursiefen
    Department of Ophthalmology, University Hospital Cologne, Cologne, Germany
  • Gereon R. Fink
    Department of Neurology, University Hospital Cologne, Cologne, Germany
  • Helmar C. Lehmann
    Department of Neurology, University Hospital Cologne, Cologne, Germany
  • Ludwig M Heindl
    Department of Ophthalmology, University Hospital Cologne, Cologne, Germany
  • Footnotes
    Commercial Relationships Franziska Bucher, None; Christian Schneider, None; Tobias Blau, None; Claus Cursiefen, None; Gereon Fink, None; Helmar Lehmann, None; Ludwig Heindl, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2015, Vol.56, 1620. doi:
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      Franziska Bucher, Christian Schneider, Tobias Blau, Claus Cursiefen, Gereon R. Fink, Helmar C. Lehmann, Ludwig M Heindl; In Vivo Confocal Microscopy in Histologically Confirmed Small Fiber Neuropathy. Invest. Ophthalmol. Vis. Sci. 2015;56(7 ):1620.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: In patients with small fiber neuropathy (SFN) non-invasive diagnostic tests that allow accurate monitoring of disease progression are urgently needed. Aim of this study was to assess corneal trigeminal small sensory nerves and immune cells by in vivo corneal confocal microscopy (CCM) in histologically confrimed SFN.

Methods: This prospective single-center study analyzed 14 patients with histologically confirmed SFN. CCM parameters (corneal nerve fiber density (NFD), total number of nerves, number of main trunks, and branches, nerve tortuosity, and dendritic cell density (DCD)) were compared to 14 age-matched healthy controls and correlated with clinical symptoms, disease course and histopathological findings.

Results: Corneal NFD (15489.3 ± 5927.6 μm/mm2 vs. 22687.1 ± 4328.7 μm/mm2; p=0.001) and total number of nerves (10.4 ± 4.6 /frame vs. 18.5 ± 4.8 /frame; p<0.0001) were significantly reduced in patients with SFN. In contrast, nerve tortuosity was significantly increased (2.2 ± 0.31 vs. 1.7 ± 0.5; p=0.02). Corneal NFD did not correlate with intraepidermal NFD (ρ -0.158; p>0.05) or clinical measures (p>0.05). Average dendritic cell density (DCD) was increased in SFN (33.5 ± 57.5 cells/mm2 vs. 16.1 ± 13.7 cells/mm2), but did not reach significance (p>0.05).

Conclusions: CCM provides parameters that reliably indicate injury to sensory afferents of the trigeminal nerve in patients with SFN. Our data suggest that CCM may serve both as a non-invasive diagnostic test and a surrogate marker in SFN.

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