June 2015
Volume 56, Issue 7
Free
ARVO Annual Meeting Abstract  |   June 2015
Neuronal cell loss in an autoimmune glaucoma model after immunization with GDNF and HSP27
Author Affiliations & Notes
  • Christina Casola
    Eye Hospital, Experimental Eye Research Institute, Bochum, Germany
  • Sabrina Reinehr
    Eye Hospital, Experimental Eye Research Institute, Bochum, Germany
  • Sandra Kuehn
    Eye Hospital, Experimental Eye Research Institute, Bochum, Germany
  • Bernhard Spiess
    Equine Department,, Vetsuisse Faculty, University of Zurich, Zurich, Switzerland
  • Burkhard Dick
    Eye Hospital, Experimental Eye Research Institute, Bochum, Germany
  • Stephanie C Joachim
    Eye Hospital, Experimental Eye Research Institute, Bochum, Germany
  • Footnotes
    Commercial Relationships Christina Casola, None; Sabrina Reinehr, None; Sandra Kuehn, None; Bernhard Spiess, None; Burkhard Dick, None; Stephanie Joachim, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2015, Vol.56, 2459. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Christina Casola, Sabrina Reinehr, Sandra Kuehn, Bernhard Spiess, Burkhard Dick, Stephanie C Joachim; Neuronal cell loss in an autoimmune glaucoma model after immunization with GDNF and HSP27. Invest. Ophthalmol. Vis. Sci. 2015;56(7 ):2459.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: Glaucoma is a vision threatening disease. Different possible pathogenic factors have been described, including the involvement of the immune system. Previously, immunization with glaucoma related antigens, like heat shock proteins (HSP), induced retinal ganglion cell (RGC) degeneration. The purpose of this study was to investigate RGC death and alterations in macroglia, amacrine cells and photoreceptors after immunization with glial cell-derived neurotrophic factor (GDNF) or GDNF in combination with HSP27.

Methods: Rats were immunized with GDNF and GDNF+HSP27. Control animals (Co) received sodium chloride (n=4-5/group). After 4 weeks cross-sections of the retina were stained with Brn-3a and NeuN to quantify RGC density. GFAP and vimentin staining was used to investigate macroglia and was confirmed by Western Blot. Amacrine cells were labelled with parvalbumin and photoreceptors with rhodopsin and opsin. Cell counts and staining area analysis were done using ImageJ software. Statistical analysis was performed using Student’s t-test.

Results: Retinas of immunized animals showed a significant loss of Brn-3a+ RGCs (Co: 29.72±4.94 cells/mm; GDNF: 20.01±6.2 cells/mm; p=0.025; GDNF+HSP27: 23.15±1.78 cells/mm; p=0.04) or NeuN+ RGCs (Co: 30.08±3.14 cells/mm; GDNF: 22.5±6.01 cells/mm; p=0.037; GDNF+HSP27: 23.22±1.54 cells/mm; p=0.005). In the GDNF group the GFAP+/vimentin+ area was significantly increased compared to Co (GFAP: p=0.00005; vimentin: p=0.007). No changes in macroglia staining could be observed in the GDNF+HSP27 group (p>0.05). A loss of amacrine cells was detected with parvalbumin staining solely in the GDNF+HSP27 group (Co: 32.91±19.41 cells/mm; GDNF: 34.26±15.18 cells/mm; p=0.55; GDNF+HSP27: 27.75±14.99 cells/mm; p=0.04). Photoreceptors were not affected in both groups.

Conclusions: Immunization with GDNF as well as the combination of GDNF+HSP27 led to a significant RGC loss. In the GDNF group an activation of macroglia was observed, but no further damage of retinal cells. In contrary, we detected no macroglia activation in the GDNF+HSP27 group, but a loss of amacrine cells. We propose, that an inhibition of glia cell activity after immunization with GDNF in combination with HSP27 occurs. Therefore, the deeper retinal layers are more affected.

×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×