June 2015
Volume 56, Issue 7
Free
ARVO Annual Meeting Abstract  |   June 2015
Alterations In Lens Fiber Structure After Cytokine Treatment Mimic Diabetic Lens Changes
Author Affiliations & Notes
  • Kristin J Al-Ghoul
    Anatomy & Cell Biology, Rush University Medical Center, Chicago, IL
  • Kevin P Khoury
    Anatomy & Cell Biology, Rush University Medical Center, Chicago, IL
  • Fareeha Mahmood
    Anatomy & Cell Biology, Rush University Medical Center, Chicago, IL
  • Footnotes
    Commercial Relationships Kristin Al-Ghoul, None; Kevin Khoury, None; Fareeha Mahmood, None
  • Footnotes
    Support None
Investigative Ophthalmology & Visual Science June 2015, Vol.56, 2633. doi:
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    • Get Citation

      Kristin J Al-Ghoul, Kevin P Khoury, Fareeha Mahmood; Alterations In Lens Fiber Structure After Cytokine Treatment Mimic Diabetic Lens Changes. Invest. Ophthalmol. Vis. Sci. 2015;56(7 ):2633.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: Our prior studies have shown that selected cytokines are elevated in the eye during diabetic cataract formation in a streptozotocin (STZ)-induced diabetic rat model. The objective of this ongoing study is to assess the direct effects of the cytokines IL-1α and IL-4 on elongating lens fiber organization and ultrastructure in a whole-lens culture model.

Methods: Normal Wistar rat lenses (n=24 animals) were cultured with either IL-1α or IL-4 at the same concentrations measured in STZ-diabetic rat eyes, or were untreated (controls). Following either 24 hours (OD) or 48 hours (OS) in culture, lenses were photographed, decapsulated and fixed for scanning electron microscopic (SEM) analysis.

Results: Cytokine-treated lenses showed gross structural alterations such as sutural widening, discrete superficial opacities and foci of fiber disruption. Changes manifested as early as 24 hours and were generally more pronounced at 48 hours. Lenses cultured in media without cytokine remained transparent and lacked obvious structural alterations. SEM examination of cytokine-treated lenses revealed regions having a disorganized pattern of swirled fiber ends and areas of elongated ends which often displayed excessively long, disorganized filopodia. In addition, zones of cellular breakdown were present in locations corresponding to gross defects.

Conclusions: The gross and ultrastructural changes that were noted after cytokine-treatment of normal lenses were consistent with documented changes in STZ-induced diabetic cataracts. These findings support the hypothesis that IL-1α and IL-4 may be involved in the initiation of diabetic cataract formation.

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