April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
AAV Vectors With Engineered Capsid Mutations Efficiently Transduce Outer Retina With Intravitreal Delivery And Result In Rapid Gene Expression With Subretinal Delivery In Dogs
Author Affiliations & Notes
  • Joshua T. Bartoe
    Small Animal Clinical Sciences, Michigan State University, East Lansing, Michigan
  • Freya Mowat
    Small Animal Clinical Sciences, Michigan State University, East Lansing, Michigan
  • Astra Dinculescu
    Dept of Ophthalmology, Univ of Florida Coll of Medicine, Gainesville, Florida
  • Sanford Boye
    Dept of Ophthalmology, Univ of Florida Coll of Medicine, Gainesville, Florida
  • William Hauswirth
    Dept of Ophthalmology, Univ of Florida Coll of Medicine, Gainesville, Florida
  • Simon Petersen-Jones
    Small Animal Clinical Sciences, Michigan State University, East Lansing, Michigan
  • Footnotes
    Commercial Relationships  Joshua T. Bartoe, None; Freya Mowat, None; Astra Dinculescu, None; Sanford Boye, None; William Hauswirth, AGTC, Inc. (P); Simon Petersen-Jones, None
  • Footnotes
    Support  Michigan State University Endowed Research Funds
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 1420. doi:
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      Joshua T. Bartoe, Freya Mowat, Astra Dinculescu, Sanford Boye, William Hauswirth, Simon Petersen-Jones; AAV Vectors With Engineered Capsid Mutations Efficiently Transduce Outer Retina With Intravitreal Delivery And Result In Rapid Gene Expression With Subretinal Delivery In Dogs. Invest. Ophthalmol. Vis. Sci. 2011;52(14):1420.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To compare transduction onset time and cellular tropism of GFP-expressing AAV vectors with Try-to-Phe capsid mutations compared with self-complimentary AAV vectors following either intravitreal or subretinal injection in canine eyes.

Methods: : 12 normal beagle dogs (24 eyes) were used. All AAV genomes included eGFP driven by the chicken beta actin promoter. Vectors were tittered and diluted to 5x1011 vg/ml. 4 right eyes (OD) received intravitreal AAV serotype 2/2 with 4 Tyr-to-Phe capsid mutations (mutAAV2); 4 contralateral left eyes (OS) received intravitreal AAV serotype 2/2 with a self-complimentary genome (scAAV2). Following 3-port pars plana vitrectomy, 4 OD received subretinal mutAAV2; 4 OS received self-complimentary AAV serotype 2/5 (scAAV5). Following vitrectomy, the remaining 4 OD received subretinal AAV serotype 2/8 with 1 Tyr-to-Phe capsid mutation (mutAAV8); 4 OS received scAAV5. Ophthalmic examination and color and fluorescent fundus photography were performed regularly. Fundic images were identically computer enhanced to detect low-level fluorescence. Eyes were fixed following euthanasia at 28 days post-injection. Retinal cryosections were evaluated for GFP. Contralateral eyes were compared statistically via paired t-test.

Results: : Onset of GFP expression was detected in all dogs with intravitreal injection of mutAAV2 by 9 days and scAAV2 by 8 days; fluorescence was similar for both vectors throughout the study. GFP expression was detected in all dogs with subretinal injection of mutAAV2 and mutAAV8 by 3 days and scAAV5 by 5 days. However, fluroescence was greater for mutAAV8 compared to both mutAAV2 and scAAV5. Histology showed no difference in transduction of inner retinal cells between intravitreal mutAAV2 and scAAV2; however, mutAAV2 more efficiently transduced the outer retina and RPE. All subretinal injected vectors efficiently transduced outer retina and RPE.

Conclusions: : AAV 2/2 vectors with engineered capsid mutations are efficient in transduction of outer retina following intravitreal delivery. AAV 2/2 and 2/8 vectors with engineered capsid mutations produce more rapid and robust gene expression following subretinal delivery. AAV vectors with engineered capsid mutations are likely to be a critical resource in gene replacement therapy for rapidly progressive heritable retinal degenerations.

Keywords: retina • gene transfer/gene therapy • retinal degenerations: hereditary 
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