April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
The Possible Mechanism of Fractalkine/CX3CR1 Mediating Microglia-Photoreceptor Crosstalk in Light-Induced Photoreceptor Degeneration
Author Affiliations & Notes
  • Zhang Meng
    Department of Ophthalmology, Eye and ENT Hospital, Fudan University, Shanghai, China
  • Liu Wei
    Department of Ophthalmology, Eye and ENT Hospital, Fudan University, Shanghai, China
  • Ni Yingqin
    Department of Ophthalmology, Eye and ENT Hospital, Fudan University, Shanghai, China
  • Xu Gezhi
    Department of Ophthalmology, Eye and ENT Hospital, Fudan University, Shanghai, China
  • Footnotes
    Commercial Relationships  Zhang Meng, None; Liu Wei, None; Ni Yingqin, None; Xu Gezhi, None
  • Footnotes
    Support  the National Basic Research Program of China 2007CB512205
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 1857. doi:
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      Zhang Meng, Liu Wei, Ni Yingqin, Xu Gezhi; The Possible Mechanism of Fractalkine/CX3CR1 Mediating Microglia-Photoreceptor Crosstalk in Light-Induced Photoreceptor Degeneration. Invest. Ophthalmol. Vis. Sci. 2011;52(14):1857.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose:
 

To investigate the role of Fractalkine/CX3CR1 in light-induced photoreceptor degeneration and the relationship between photoreceptor apoptosis and retinal microglial activation.

 
Methods:
 

Sprague-Dawley rats were exposed to intense blue light for 24 hours. Antibodies were used to label Fractalkine/CX3CR1 and retinal microglia at different time points. The expression of Fractalkine/CX3CR1 was detected by Real Time-PCR and Western immunoblot analysis. TUNEL method was used to detect cell apoptosis in retina. MAPKs were also examined to evaluate the possible signal pathway of microglia-photoreceptor crosstalk.

 
Results:
 

Our results showed the expression of Fractalkine increased in the outer nuclear layer of the retina and OX42-positive microglia also occurred in the same place. CX3CR1 was expressed major in the microglial. Expression of Fractalkine/CX3CR1 mRNA and protein were also significantly increased after explosion to light.

 
Conclusions:
 

These findings demonstrate that Fractalkine/CX3CR1 may play an important role in the microglia-photoreceptor crosstalk in Light-induced photoreceptor degeneration.  

 
Keywords: microglia • retinal degenerations: cell biology • neuroprotection 
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