March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Th17 Cell-associated Chemokine Receptor and Ligand Expression in Dry Eye Disease
Author Affiliations & Notes
  • Thomas H. Dohlman
    Schepens Eye Research Institute, Harvard Medical School, Boston, Massachusetts
  • Shilpa Kodati
    Schepens Eye Research Institute, Harvard Medical School, Boston, Massachusetts
  • Jing Hua
    Schepens Eye Research Institute, Harvard Medical School, Boston, Massachusetts
  • Yihe Chen
    Schepens Eye Research Institute, Harvard Medical School, Boston, Massachusetts
  • Sunil K. Chauhan
    Schepens Eye Research Institute, Harvard Medical School, Boston, Massachusetts
  • Reza Dana
    Schepens Eye Research Institute, Harvard Medical School, Boston, Massachusetts
  • Footnotes
    Commercial Relationships  Thomas H. Dohlman, None; Shilpa Kodati, None; Jing Hua, None; Yihe Chen, None; Sunil K. Chauhan, None; Reza Dana, None
  • Footnotes
    Support  NIH EY20889
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 2322. doi:
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    • Get Citation

      Thomas H. Dohlman, Shilpa Kodati, Jing Hua, Yihe Chen, Sunil K. Chauhan, Reza Dana; Th17 Cell-associated Chemokine Receptor and Ligand Expression in Dry Eye Disease. Invest. Ophthalmol. Vis. Sci. 2012;53(14):2322.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : Th17 cells have been reported to be the predominate effector T cell in dry eye disease (DED). The purpose of this study was to investigate the chemokine receptor-ligand system in DED, which may be involved in Th17 cell homing to the ocular surface in this disorder.

Methods: : 8-10 week old female C57BL/6 mice were housed in a low humidity controlled environment chamber for 12 days to induce DED. Expression of the Th17 associated chemokine receptors CCR2 and CCR6 was determined by flow cytometric analysis of Th17 (CD4+IL-17+) cells collected from the draining lymph nodes (LN). Conjunctival samples were analyzed via real time PCR to determine expression of the chemokines CCL2 and CCL20.

Results: : Analysis of the draining LN of DED mice showed a 50% increase in the frequency of CCR2-hi expressing Th17 cells and a 30% increase in CCR6 expression by all Th17 cells compared to normal mice. In addition, the conjunctiva of DED mice showed a significant 12 fold increase in CCL20 (p<0.05) expression, and a 2 fold increase in CCL2 expression compared to conjunctiva from normal mice.

Conclusions: : Our data demonstrate increased CCR2 and CCR6 expression by Th17 cells and increased levels of corresponding chemokine ligands at the ocular surface in DED. These changes may represent a mechanism for Th17 cell migration to the ocular surface in DED.

Keywords: cornea: tears/tear film/dry eye • inflammation 
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