April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
Cyclosporin A Suppresses Tgfβ1-induced Collagen Typei/iv Expression In Both Primary And Cell Line Of Conjunctival Fibroblasts: A Possible Implication In Modulation Of Fibrosis
Author Affiliations & Notes
  • Alessandra Micera
    Lab. Ophthalmology, IRCCS-GB Bietti Eye Foundation, Rome, Italy
    CIR, Lab Ophthalmology, University Campus Bio-Medico, Rome, Italy
  • Bijorn Omar Balzamino
    Lab. Ophthalmology, IRCCS-GB Bietti Eye Foundation, Rome, Italy
    CIR, Lab Ophthalmology, University Campus Bio-Medico, Rome, Italy
  • Loredana Mastrella
    CIR, Lab Ophthalmology, University Campus Bio-Medico, Rome, Italy
  • Paolo Lauretti
    CIR, Lab Ophthalmology, University Campus Bio-Medico, Rome, Italy
  • Stefano Bonini
    CIR, Lab Ophthalmology, University Campus Bio-Medico, Rome, Italy
  • Footnotes
    Commercial Relationships  Alessandra Micera, None; Bijorn Omar Balzamino, None; Loredana Mastrella, None; Paolo Lauretti, None; Stefano Bonini, None
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 2014. doi:
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      Alessandra Micera, Bijorn Omar Balzamino, Loredana Mastrella, Paolo Lauretti, Stefano Bonini; Cyclosporin A Suppresses Tgfβ1-induced Collagen Typei/iv Expression In Both Primary And Cell Line Of Conjunctival Fibroblasts: A Possible Implication In Modulation Of Fibrosis. Invest. Ophthalmol. Vis. Sci. 2011;52(14):2014.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To explore in vitro the influence of Cyclosporin A (CsA), a powerful immunosuppressive agent, on fibrosis in term of proliferation, differentiation, ECM metabolism and contraction of primary (from n=3/biopsies and from ScienCell Res. Lab, Carlsbad, CA) cultures of conjunctival fibroblasts. CsA effects were compared with those of TGFβ1.

Methods: : Both fibroblasts (FBs) and in vitro induced myofibroblasts (myoFBs) were used for CsA (Restasis, Allergan Inc., Irvine, CA) at scalar doses (50, 200, 400 and 800 ng/mL). Cells were also exposed to 10ng/mL TGFβ1, 800ng/mL CsA either alone or combined. Sister untreated cells were used as control. After 24 hours, cell viability and proliferation were assessed. Extracellular metabolism was investigated by measuring collagen typeI/IV (csELISA) and MMP9/TIMP1 (zymography, ssELISA) protein expression. Extracellular matrix contraction was monitored according to the 3D gel assay. Statistical analysis was assessed by parametric ANOVA-Tukey Kramer post hoc assays.

Results: : Increasing CsA doses did not influence either proliferation nor αSMA expression in both FBs and myoFBs. CsA did not modulate significantly collagen typeI/IV nor MMP9 expression, while TGFβ1 exposure resulted in a drastic increase of collagen typeI/IV in both cell types (p<.05). A trend toward a MMP9 increase was detected upon CsA exposure while a trend to a decrease was observed for TIMP1 (p>.05). Addition of 800ng/mL CsA to 10ng/mL TGFβ1 abolished this typeI/IV increase in both FBs and myoFBs (p<.05). Merely to MMP9/TIMP1, addition of 800ng/mL CsA to 10ng/mL TGFβ1 resulted in a suppression of TGFβ1-induced MMP9/TIMP1 expression in both cell types. At low doses, CsA triggered FB mediated contraction of a 3D gel with no effects at higher doses, as compared to those of TGFβ1.

Conclusions: : CsA eye drops are therapeutically used for severe allergic conjunctivitis, but the mechanism of action is actually unknown. Herein, CsA appears to modulate profibrogenic effects induced by TGFβ1, a well-known factor involved in FBs/myoFBs activation, suggesting new potential mechanisms of CsA in ocular tissue remodeling/fibrosis.

Keywords: conjunctiva • extracellular matrix • cyclosporine 
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