April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
A Shared Hla-drb1 Epitope In The Dr Beta First Domain Is Associated With Vogt-koyanagi-harada Syndrome In Indian Patients
Author Affiliations & Notes
  • Edoardo Baglivo
    Clinique d'Ophtalmologie,
    Geneva University Hospital, Geneva, Switzerland
  • Rathinam Sivakumar
    Uveitis Clinic, Aravind Eye Hospital, Madurai, India
  • Marianne Gex-Fabry
    PS-Recherche Clinique,
    Geneva University Hospital, Geneva, Switzerland
  • Jean-Marie Tiercy
    National Reference Laboratory for Histocompatibility, Transplantation Immunology Unit,
    Geneva University Hospital, Geneva, Switzerland
  • Footnotes
    Commercial Relationships  Edoardo Baglivo, None; Rathinam Sivakumar, None; Marianne Gex-Fabry, None; Jean-Marie Tiercy, None
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 2384. doi:
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      Edoardo Baglivo, Rathinam Sivakumar, Marianne Gex-Fabry, Jean-Marie Tiercy; A Shared Hla-drb1 Epitope In The Dr Beta First Domain Is Associated With Vogt-koyanagi-harada Syndrome In Indian Patients. Invest. Ophthalmol. Vis. Sci. 2011;52(14):2384.

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Abstract
 
Purpose:
 

Vogt-Koyanagi-Harada (VKH) disease and sympathetic ophthalmia (SO) are two distinct entities that share common clinical and histopathological features. It is unknown whether they have a common genetic susceptibility. We present in this paper the HLA-DRB1 genotyping analysis of a large cohort of VKH patients from southern India and compare these patients to patients with SO and to healthy individuals from the same geographic area.

 
Methods:
 

VKH patients were diagnosed according to the revised criteria of the International Committee on VKH disease. Genomic DNA was extracted from all patients and controls. Samples were analyzed for HLA-DRB1 alleles by reverse polymerase chain reaction (PCR) sequence-specific oligonucleotide (SSO) hybridization on microbeads, using the Luminex technology, and by PCR sequence-specific primers (SSP) typing for DRB1*04 allele determination. Strength of associations was estimated by odds ratios (OR) and 95% confidence intervals (CI) and frequencies were compared using the Fisher's exact test.

 
Results:
 

HLA-DRB1 alleles were determined in 94 VKH patients, 39 SO patients, and 112 healthy controls. HLA-DRB1*04 frequency was higher in VKH patients (20.2% versus 10.3% in controls; OR=2.2, p=0.005, pc=0.067). This association was lower than the association of HLA-DRB1*04 frequency in cohorts of patients from different origins. No significant DR4 association with SO was detected. HLA-DRB1*0405 and HLA-DRB1*0410 alleles were significantly increased in VKH patients (8.5% versus 0.9% in controls; OR=10.3, 95% CI=2.34-45.5, p<0.001). These two alleles share the epitope S57-LLEQRRAA (67-74) in the third hypervariable region of the HLA-DR molecule. None of the DRB1 alleles was significantly associated with SO.

 
Conclusions:
 

Based on the association of HLA-DRB1*0405 and HLA-DRB1*0410 alleles with VKH disease, we propose that the epitope S57-LLEQRRAA (67-74) in the third hypervariable region of the HLA-DRbeta1 molecule is the relevant susceptibility epitope. This genetic component seems specific to VKH disease since no correlation could be identified in SO patients. The weaker association with HLA-DR4 in this VKH patient cohort compared to VKH patients from northern India is probably related to the lower frequency of HLA-DRB1*0405 in our study group.

 
Keywords: uvea • genetics • gene screening 
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