March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Generation of NADPH by Mitochondrial-Mediated Pathways in Mouse Rod Photoreceptors
Author Affiliations & Notes
  • Leopold Adler, IV
    Physics and Astronomy, College of Charleston, Charleston, South Carolina
    Ophthalmology, Medical University of South Carolina, Charleston, South Carolina
  • Chunhe Chen
    Ophthalmology, Medical University of South Carolina, Charleston, South Carolina
  • Yiannis Koutalos
    Ophthalmology, Medical University of South Carolina, Charleston, South Carolina
  • Footnotes
    Commercial Relationships  Leopold Adler, IV, None; Chunhe Chen, None; Yiannis Koutalos, None
  • Footnotes
    Support  NIH/NEI grant EY014850, and an unrestricted grant to MUSC Storm Eye Institute from Research to Prevent Blindness, Inc., New York, NY.
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 3360. doi:
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    • Get Citation

      Leopold Adler, IV, Chunhe Chen, Yiannis Koutalos; Generation of NADPH by Mitochondrial-Mediated Pathways in Mouse Rod Photoreceptors. Invest. Ophthalmol. Vis. Sci. 2012;53(14):3360.

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Abstract

Purpose: : To determine the contribution of different NADPH-generating metabolic pathways to the reduction of all-trans retinal to retinol in the outer segment of mouse rod photoreceptors. All-trans retinol is formed in rod outer segments from the reduction of the all-trans retinal released from photoactivated rhodopsin via a reaction that utilizes NADPH.

Methods: : Experiments were carried out with dark-adapted living rod photoreceptors isolated from 2-3 months old 129/sv wild type mice. The amount of all-trans retinol in the outer segment was measured at various times after bleaching from its fluorescence (excitation 360 nm; emission >420 nm). Experiments were carried out at 37 0C. A numerical model describing the formation of all-trans retinol was developed. From this model, the concentration of and production rate of NADPH were estimated from the difference in the known kinetics of all-trans retinal release and those of all-trans retinol formation.

Results: : All-trans retinol formation was suppressed in the absence of glucose, and was restored by addition of pyruvate. In the presence of 5 mM glucose, NADPH was supplied to the rod outer segment at a rate of ~0.22 mM min-1. The concentration of cytosolic outer segment NADPH present before bleaching was ~0.3 mM. In the absence of glucose, concentrations of 0.5 - 1.0 mM of pyruvate were sufficient to restore NADPH supply rates of 0.20 - 0.24 mM min-1.

Conclusions: : Mitochondrial-mediated pathways can generate NADPH at sufficient rates to sustain the reduction of all-trans retinal to retinol following rhodopsin bleaching.

Keywords: photoreceptors • metabolism • retinoids/retinoid binding proteins 
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