March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Shear stress induced multimerization of cochlin and its interaction with Annexin A2
Author Affiliations & Notes
  • Renata Picciani
    Ophthalmology, Bascom Palmer Eye Institute, Miami, Florida
  • Sanjoy K. Bhattacharya
    Ophthalmology, Bascom Palmer Eye Institute, Miami, Florida
  • Horacio M. Serra
    Bioquimica Clinica, CIBICI, Fac ultad de Cs Quimicas UNC, Cordoba, Argentina
  • Footnotes
    Commercial Relationships  Renata Picciani, None; Sanjoy K. Bhattacharya, None; Horacio M. Serra, None
  • Footnotes
    Support  RPB Career award (SKB), an unrestricted grant from RPB to University of Miami, NIH grant EY016112
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 3858. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Renata Picciani, Sanjoy K. Bhattacharya, Horacio M. Serra; Shear stress induced multimerization of cochlin and its interaction with Annexin A2. Invest. Ophthalmol. Vis. Sci. 2012;53(14):3858.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : To characterize cochlin multimerization in response to shear stress and determine the interaction of native and multimerized cochlin with Annexin A2.

Methods: : Native cochlin and recombinant cochlin domains were purified from HEK cells and E.coli. The proteins were subjected to purification using standard methods. Purified proteins were incubated with trabecular meshwork extract with or without being subjected to shear stress and their interaction with Annexin A2 was probed using immunoprecipitation and Western analyses. Cochlin-Annexin A2 interactions were also probed with overlay assay and mass spectrometry using a LCQ Deca XP instrument.

Results: : We found native cochlin to interact with Annexin A2. In our kinetic assays, multimerized cochlin interacted with slightly higher binding strength with Annexin A2. The recombinant cochlin domains (LC and vWFA domains) poorly expressed either in E.coli or in HEK cells, they also failed to fold properly upon expression.

Conclusions: : Multimeric cochlin formed in response to shear stress interacts with higher binding strength with Annexin A2 compared to native cochlin.

Keywords: protein structure/function • proteomics • trabecular meshwork 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×