March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Choroidal Thickness Increase Is Different among Angle-Closure Versus Open-Angle Eyes but Does Not Explain IOP Rise after Water Drinking
Author Affiliations & Notes
  • Karun S. Arora
    Ophthalmology, Johns Hopkins University, Baltimore, Maryland
  • Joan L. Jefferys
    Ophthalmology, Johns Hopkins University, Baltimore, Maryland
  • Eugenio A. Maul
    Ophthalmology, Johns Hopkins University, Baltimore, Maryland
  • Harry A. Quigley
    Ophthalmology, Johns Hopkins University, Baltimore, Maryland
  • Footnotes
    Commercial Relationships  Karun S. Arora, None; Joan L. Jefferys, None; Eugenio A. Maul, None; Harry A. Quigley, Alcon (C), Genentech (C), Merck (C), Ono (C), Zeiss (C)
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 4173. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Karun S. Arora, Joan L. Jefferys, Eugenio A. Maul, Harry A. Quigley; Choroidal Thickness Increase Is Different among Angle-Closure Versus Open-Angle Eyes but Does Not Explain IOP Rise after Water Drinking. Invest. Ophthalmol. Vis. Sci. 2012;53(14):4173.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract
 
Purpose:
 

1) To measure potential change in choroidal thickness (CT) as a cause of intraocular pressure (IOP) rise during the water-drinking test (WDT); 2) To compare WDT responses in persons with open angle glaucoma (OAG), OAG suspects (OAGS), angle closure (AC), AC suspects (ACS) and AC glaucoma (ACG).

 
Methods:
 

Spectral domain optical coherence tomography scans of the macula with enhanced depth imaging were obtained (one eye per subject) before and 30 minutes after drinking 1 liter of water, in 25 ACS/AC/ACG and 48 OAGS/OAG subjects. Standard demographics, degree of field loss, and blood pressure were measured (before and after WDT). Average CT (6 mm scan centered on fovea) was measured with Image J software, and univariable and multivariable statistics were performed.

 
Results:
 

Baseline CT was thicker in ACG than OAG eyes (312 vs. 244µm, p=0.03). However, CT has been shown to be inversely related to axial length (AL), and OAG eyes had longer AL than ACG eyes (24.3 vs. 23.0 mm, p<0.0001), possibly accounting for this difference. After WD, mean IOP rose 40% (5.51 + 3.51 mm, p<0.0001), and systolic, mean and diastolic blood pressures (BP) also rose (8.9, 6.4, and 5.1 mm Hg, respectively, all p<0.0001), as did systolic perfusion pressure (PP, 3.4 mm Hg, p=0.01). The AL also increased (0.012 mm, p=<0.0001), though the increase was not significantly different between OA and AC eyes (0.009 mm vs. 0.017 mm, p=0.19). The observed increase in AL, most likely associated with an increase in eye volume caused by the increase in IOP, matched the expected increase in AL calculated using the pressure-volume relationship for the eye described previously (Silver and Geyer, Curr Eye Res 2000). Mean CT increased, but insignificantly (1.4 + 16.3 µm, p=0.46). Overall IOP change was not significantly associated with CT change (p=0.8). CT change was unrelated to age, sex, gender, axial length, visual field defect level, baseline IOP or baseline CT. In multivariable models in 64 eyes with >2 mm IOP rise, CT change was significantly related to change in mean or diastolic PP (p=0.02, 0.01), and AC eyes had greater change in CT than OA eyes (p=0.03).

 
Conclusions:
 

CT is responsive to PP, yet IOP increase in WDT is associated with BP rise but not with CT increase, suggesting it results from changed aqueous in/outflow related to adrenergic stimulation. Our results confirm thicker choroid in AC eyes and differences in CT reactivity in AC versus OA eyes.

 
Keywords: choroid • clinical (human) or epidemiologic studies: risk factor assessment • intraocular pressure 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×