April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
Longitudinal Study of Retinal Angiomatosis and Visual Function in Ocular von Hippel-Lindau (VHL) Disease
Author Affiliations & Notes
  • Brian C. Toy
    National Eye Institute, National Institutes of Health, Bethesda, Maryland
  • Elvira Agrón
    National Eye Institute, National Institutes of Health, Bethesda, Maryland
  • Divya Nigam
    National Eye Institute, National Institutes of Health, Bethesda, Maryland
  • Emily Y. Chew
    National Eye Institute, National Institutes of Health, Bethesda, Maryland
  • Wai T. Wong
    National Eye Institute, National Institutes of Health, Bethesda, Maryland
  • Footnotes
    Commercial Relationships  Brian C. Toy, None; Elvira Agrón, None; Divya Nigam, None; Emily Y. Chew, None; Wai T. Wong, None
  • Footnotes
    Support  NEI Intramural Research Program, and BT is supported by the Clinical Research Training Program, a public-private partnership supported jointly by the NIH and Pfizer Inc.
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 4544. doi:
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      Brian C. Toy, Elvira Agrón, Divya Nigam, Emily Y. Chew, Wai T. Wong; Longitudinal Study of Retinal Angiomatosis and Visual Function in Ocular von Hippel-Lindau (VHL) Disease. Invest. Ophthalmol. Vis. Sci. 2011;52(14):4544.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : We aimed to characterize the long-term anatomic and visual function changes associated with ocular VHL disease in patients examined at the National Eye Institute/National Institutes of Health. The results of this study may be useful in providing clinical guidance for the management of persons with ocular VHL disease.

Methods: : Patients with clinical or genetic evidence of VHL disease and at least 2 years of ophthalmic follow-up were identified and their ophthalmic records retrospectively analyzed. Univariate statistical analysis and logistic regression were performed using custom Matlab scripts and SAS.

Results: : Of 249 patients at study baseline, 154 (62%) had evidence of ocular VHL in at least one eye (29% unilateral, 33% bilateral). Of 498 eyes analyzed, 236 eyes (47%) were affected by VHL disease at baseline. Longitudinal analyses over a mean follow up period of 8.2±4.0 years revealed that 50 out of 167 at-risk participants (30%) developed new ocular VHL in a previously unaffected eye during follow-up. Of 262 eyes without ocular involvement at baseline, 58 eyes (22%) developed new ocular involvement. In eyes with existing ocular involvement at baseline, 6% developed angiomas in a new retinal location (juxtapapillary or peripheral), 20% increased in peripheral angioma number (from ≤3 to >3), 19% increased in the extent of peripheral retinal involvement (from ≤1 to >1 quadrant), and 7% progressed to end-stage disease. Significant decreases in mean ETDRS visual acuity were found in eyes developing angiomas in a new retinal location (-6.1 letters; p=0.04, paired t-test), developing new juxtapapillary angiomas (-11.4 letters; p=0.005), developing new peripheral angiomas (-4.3 letters; p=0.02), increasing in peripheral angioma number (-12.5 letters; p=0.002), or increasing in peripheral involvement (-7.6 letters; p=0.004).

Conclusions: : The overall rate of developing new ocular VHL disease in patients with VHL disease was relatively slow. A majority of eyes that were without ocular VHL at baseline did not acquire ocular involvement during follow-up. Eyes with existing ocular VHL disease also demonstrated relative stability with a minority of eyes showing increases in measures of angioma burden during follow up. Increasing angioma burden, particularly the development of new juxtapapillary angiomas, was associated with a significant long-term worsening of visual acuity.

Keywords: tumors • retina • clinical (human) or epidemiologic studies: natural history 
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