March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Autocrine protein expression of cells from the ocular surface after treatment with Plasma Rich in Growth Factors (PRGF-Endoret)
Author Affiliations & Notes
  • Francisco Jose Muruzabal
    Biotechnology Institute, Vitoria, Spain
  • Maria de la Fuente
    Biotechnology Institute, Vitoria, Spain
  • Gorka Orive
    Biotechnology Institute, Vitoria, Spain
  • Eduardo Anitua
    Biotechnology Institute, Vitoria, Spain
  • Footnotes
    Commercial Relationships  Francisco Jose Muruzabal, I'm a scientist at Biotechnology Institute (a biotechnological company) (E); Maria de la Fuente, I'm a scientist at Biotechnology Institute (a biotechnological company) (E); Gorka Orive, I'm a scientist at Biotechnology Institute (a biotechnological company) (E); Eduardo Anitua, I'm a scientist at Biotechnology Institute (a biotechnological company) (E)
  • Footnotes
    Support  Centre for Industrial Technological Development (CDTI) from Spanish government
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 4748. doi:
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      Francisco Jose Muruzabal, Maria de la Fuente, Gorka Orive, Eduardo Anitua; Autocrine protein expression of cells from the ocular surface after treatment with Plasma Rich in Growth Factors (PRGF-Endoret). Invest. Ophthalmol. Vis. Sci. 2012;53(14):4748.

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Abstract

Purpose: : To evaluate the autocrine protein expression of cells from the ocular surface after treatment with autologous PRGF-Endoret.

Methods: : Blood from healthy donors was collected, centrifuged and Plasma Rich in Growth Factors (PRGF-Endoret) was prepared avoiding the buffy coat. Human cultured cells including keratocytes (HCK), conjunctival fibroblasts (HconF) and immortalized corneal epithelial cells (HCE) were treated 48h or 72h with PRGF-Endoret. Cell culture supernatants (conditioned media) were collected at those times. Enzyme-Linked ImmunoSorbent Assay (ELISAs) method was used to measured in conditioned media several factors involved in different regeneration processes like proliferation, migration, angiogenesis or extracellular matrix remodeling.

Results: : PRGF-Endoret treatment of all types cells induced an increase of anti-angiogenesis factors like trombospondin-1, and a reduction of some angiogenic factors like VEGF; showing a possible regulatory effect of corneal angiogenesis. Keratocytes and conjunctival fibroblasts showed an increased expression of metallopeptidase inhibitor 1 (TIMP-1), hyaluronic acid and procollagen after PRGF-Endoret treatment, indicating a possible regulatory effect of extracellular matrix remodeling. The increased expression of HGF by HConF and HCK cells after PRGF-Endoret stimulation reveals a potential paracrine stimulation for corneal re-epithelialization.

Conclusions: : The PRGF-Endoret stimulates significantly the autocrine expression of several factors involved in ocular surface tissue regeneration.

Keywords: wound healing • growth factors/growth factor receptors • cornea: stroma and keratocytes 
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