March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Effects of Memantine on the mitochondrial membrane potential (m) and Lactate Dehydrogenase (LDH) production on ARPE-19 and Müller cells (MIO-M1) exposed to Hydroquinone (HQ) In Vitro
Author Affiliations & Notes
  • Claudio A. Ramirez
    Gavin Herbert Eye Institute Ophthalmology, University of California, Irvine, California
  • Mohamed Tarek
    Gavin Herbert Eye Institute Ophthalmology, University of California, Irvine, California
  • Khoa D. Pham
    Gavin Herbert Eye Institute Ophthalmology, University of California, Irvine, California
  • Marilyn Chwa
    Gavin Herbert Eye Institute Ophthalmology, University of California, Irvine, California
  • Astrid Limb
    Department of Pathology and Cell Biology, University College, London, United Kingdom
  • Baruch D. Kuppermann
    Gavin Herbert Eye Institute Ophthalmology, University of California, Irvine, California
  • M C. Kenney
    Gavin Herbert Eye Institute Ophthalmology, University of California, Irvine, California
  • Footnotes
    Commercial Relationships  Claudio A. Ramirez, None; Mohamed Tarek, None; Khoa D. Pham, None; Marilyn Chwa, None; Astrid Limb, None; Baruch D. Kuppermann, None; M. C. Kenney, None
  • Footnotes
    Support  Supported by Discovery Eye Foundation, Polly & Michael Smith Foundation, Beckman Initiative for Macular Research, Lincy Foundation, Iris & B. Gerald Cantor Foundation, Research to Prevent Blindness
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 4766. doi:
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      Claudio A. Ramirez, Mohamed Tarek, Khoa D. Pham, Marilyn Chwa, Astrid Limb, Baruch D. Kuppermann, M C. Kenney; Effects of Memantine on the mitochondrial membrane potential (m) and Lactate Dehydrogenase (LDH) production on ARPE-19 and Müller cells (MIO-M1) exposed to Hydroquinone (HQ) In Vitro. Invest. Ophthalmol. Vis. Sci. 2012;53(14):4766.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To study the in vitro effects of memantine, a neuroptotectant used in patients with Alzheimer’s disease and glaucoma, on human ARPE-19 and MIO-M1 cells exposed to hydroquinone (HQ), one of the toxic components of cigarette smoke

Methods: : ARPE-19 and MIO-M1 cells were pretreated for 6 hours with 30µM of memantine and then exposed to 200µM, 100µM, 50µM and 25µM of HQ (dissolved in DMSO), while the memantine was still present. JC1 assay was performed to evaluate the mitochondrial membrane potential (ΔΨm). LDH production was measured by the LDH-Cytotoxicity Assay Kit II.

Results: : The ΔΨm for ARPE-19 cells exposed to 200µM, 100µM, 50µM and 25µM of HQ was 3770±43.08, 4015±66.51, 5139±40.76 and 7475±94.06, respectively compared to the highest (200 µM) DMSO equivalent-treated cells (9097±76.06). Pretreatment with 30µM memantine resulted in an increased ΔΨm only at the 50 µM HQ dose, at the level of 6997±75.45 (p<0.0001). The values for memantine pretreated cultures plus 200µM, 100µM and 25µM HQ were 3777±23.23(p=0.8787), 4181±39.52(p =0.0573) and 7637±41.71(p =0.1461), respectively. ΔΨm for MIO-M1 treated cultures plus 200µM, 100µM, 50µM and 25µM HQ were 3275±81.50, 4527±61.49, 5625±57.80 and 7763±34.77, respectively compared to the highest (200 µM) DMSO equivalent -treated cells (9118±45.9). In the 50µM HQ dose, after memantine pretreatment the ΔΨm increased to 6695±34.03(p <0.0001 ). There were no statistically significant differences for the other HQ doses. LDH levels for ARPE-19 cells treated with 200µM, 100µM, 50µM and 25µM HQ were 5.50±0.25, 4.8±0.20, 4.16±0.06 and 2.0±0.10 . The LDH levels were decreased significantly after pretreated with memantine at 50 µM and 25 µM HQ doses to 2.233±0.08(p <0.0001) and 1.567±0.12(p< 0.0314). MIO-M1 cells treated with the same concentrations of HQ alone showed LDH levels of 6.20±0.11, 5.66±0.08, 5.35±0.13 and 2.06±0.03. The level of LDH decreased significantly after pretreatment with memantine only at the 25 µM HQ to 1.250±0.08 (p< 0.0007).

Conclusions: : Memantine showed an in vitro protective effect against HQ induced toxicity by increasing the ΔΨm at 50µM HQ dose on both cell lines and decreasing LDH levels at 50µM and 25µM HQ doses in ARPE-19 cells and at the 25µM HQ dose on MIO-M1 cells.

Keywords: neuroprotection • retinal culture • Muller cells 
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