April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
Autofluorescence Study In NR2E3 p.G56R-linked Autosomal Dominant Retinitis Pigmentosa In A Single Kindred
Author Affiliations & Notes
  • Hoai V. Tran
    Jules-Gonin Eye Hospital, Lausanne, Switzerland
  • Pascal Escher
    IRO-Institute for Research in Ophthalmology, Sion, Switzerland
  • Veronika Vaclavik
    Jules-Gonin Eye Hospital, Lausanne, Switzerland
  • Isabelle Favre
    IRO-Institute for Research in Ophthalmology, Sion, Switzerland
  • Hasret Bajrami
    IRO-Institute for Research in Ophthalmology, Sion, Switzerland
  • Francois X. Borruat
    Jules-Gonin Eye Hospital, Lausanne, Switzerland
  • Daniel F. Schorderet
    IRO-Institute for Research in Ophthalmology, Sion, Switzerland
  • Francis L. Munier
    Jules-Gonin Eye Hospital, Lausanne, Switzerland
  • Footnotes
    Commercial Relationships  Hoai V. Tran, None; Pascal Escher, None; Veronika Vaclavik, None; Isabelle Favre, None; Hasret Bajrami, None; Francois X. Borruat, None; Daniel F. Schorderet, None; Francis L. Munier, None
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 6604. doi:
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      Hoai V. Tran, Pascal Escher, Veronika Vaclavik, Isabelle Favre, Hasret Bajrami, Francois X. Borruat, Daniel F. Schorderet, Francis L. Munier; Autofluorescence Study In NR2E3 p.G56R-linked Autosomal Dominant Retinitis Pigmentosa In A Single Kindred. Invest. Ophthalmol. Vis. Sci. 2011;52(14):6604.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To date, the genotype/phenotype correlation of p.G56R-linked autosomal dominant retinitis pigmentosa (ADRP) is limited to less than 10 kindred. The purpose of this study is to report an unusual appearance of fundus autofluorescence (AF) with NR2E3 p.G56R-linked ADRP in a single kindred.

Methods: : Patients were enrolled among three generations in a previously unreported family. Molecular diagnosis was performed on all exons of NR2E3 and a p.G56R mutation was identified in affected family members only. Examinations included fundus photography, visual fields, optical coherence tomography, AF, near-infrared AF and ISCEV-standard electrophysiology (ERG).

Results: : Among 10 examined family members, 5 were affected. The youngest and oldest patients were 16 and 65 years old, respectively. Fundus examination revealed a range of retinal disorder from normal to optic nerve pallor, attenuated arterial caliber and bone spicule-like pigment deposits. In all patients, AF showed a double hyperfluorescent ring; an inner paramacular ring which extension was comparable among patients and an outer ring along the vascular arcades which extended towards periphery in older patients and became hypofluorescent. Maximal scotopic ERGs when recordable showed an increased a/b wave ratio.

Conclusions: : A double hyperfluorescent ring on AF is an uncommon observation and might be a specific clinical finding in NR2E3 p.G56R-linked ADRP. The consistency of that finding in all affected members of our 3-generation family confirms a previous study. Further analysis is required to determine whether AF changes are associated with particular retinal layer abnormalities.

Keywords: retinal degenerations: hereditary • imaging/image analysis: clinical • genetics 
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