March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Cytotoxicity of Ganciclovir on Cultured Human Corneal Endothelial Cells
Author Affiliations & Notes
  • Young Joo Shin
    Ophthalmology, Hallym University College of Medicine, Seoul, Republic of Korea
  • Jae Woong Koh
    Ophthalmology, Chosun University School of Medicine, Kwangju, Republic of Korea
  • Tae Young Chung
    Ophthalmology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Republic of Korea
  • Joon Young Hyon
    Ophthalmology, Seoul National University College of Medicine, Seoul, Republic of Korea
  • Won Ryang Wee
    Ophthalmology, Seoul National University College of Medicine, Seoul, Republic of Korea
  • Footnotes
    Commercial Relationships  Young Joo Shin, None; Jae Woong Koh, None; Tae Young Chung, None; Joon Young Hyon, None; Won Ryang Wee, None
  • Footnotes
    Support  This study was supported by the Korea Science and Engineering Foundation (KOSEF) grant (2010-0021571) funded by the Korea government (MEST) and by a grant from Hallym University Medical Center Researc
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 6020. doi:
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      Young Joo Shin, Jae Woong Koh, Tae Young Chung, Joon Young Hyon, Won Ryang Wee; Cytotoxicity of Ganciclovir on Cultured Human Corneal Endothelial Cells. Invest. Ophthalmol. Vis. Sci. 2012;53(14):6020.

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Abstract

Purpose: : The principal objective of this study is to evaluate the cytotoxic effect of ganciclovir (GCV) on the cultured human corneal endothelial cells (HCECs).

Methods: : HCECs were cultured according to a previously published method and exposed to various concentrations of (0 - 20 mg/ml) of GCV. Cell viability was assessed via the Cell Counting Kit-8 method and live/dead viability/cytotoxicity assays. Cell damage was assessed using phase-contrast microscopy after 24 hr exposure to GCV. Cell cycle and apoptotic effects were analyzed by NC-3000 to evaluate the effects of GCV on HCECs.

Results: : Cytotoxicity tests demonstrated the cytotoxic effect of GCV on HCECs in a dose dependent manner. GCV concentrations of ≥ 5 mg/mL led to a significant reduction in cell viability. Higher concentrations of GCV resulted in an increased number of apoptotic cells indicating activation of the pro-apoptotic pathway.

Conclusions: : GCV has a dose-dependent toxic effect on cultured HCECs. Our results suggest that intracameral GCV concentrations of ≥ 5 mg/mL may increase the risk of corneal endothelial damage although GCV concentrations of up to 0.5 mg/mL do not decrease cell viability.

Keywords: antiviral drugs • cornea: endothelium • drug toxicity/drug effects 
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