March 2012
Volume 53, Issue 14
Free
ARVO Annual Meeting Abstract  |   March 2012
Corneal Dendritic Cells Suppress Local Corneal Damage and Mediate Systemic Viral Dissemination in Herpes Simplex Keratitis
Author Affiliations & Notes
  • Kai Hu
    Ophthalmology, Massachusetts Eye & Ear Infirmary, Boston, Massachusetts
  • Homayon Ghiasi
    Surgery/Ophthal Research, Cedars-Sinai Medical Center, Los Angeles, California
  • Ulrich H. Von Andrian
    Immune Disease institute, Boston, Massachusetts
  • Pedram Hamrah
    Ophthalmology, Massachusetts Eye & Ear Infirmary, Boston, Massachusetts
    Immue Institute Disease, Boston, Massachusetts
  • Footnotes
    Commercial Relationships  Kai Hu, None; Homayon Ghiasi, None; Ulrich H. Von Andrian, None; Pedram Hamrah, None
  • Footnotes
    Support  NIH K08-EY020575, MEEI Foundation, RPB Career Development Award
Investigative Ophthalmology & Visual Science March 2012, Vol.53, 6154. doi:
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      Kai Hu, Homayon Ghiasi, Ulrich H. Von Andrian, Pedram Hamrah; Corneal Dendritic Cells Suppress Local Corneal Damage and Mediate Systemic Viral Dissemination in Herpes Simplex Keratitis. Invest. Ophthalmol. Vis. Sci. 2012;53(14):6154.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : Corneal antigen-presenting cells (APCs) play an important role in infectious keratitis. We recently demonstrated increased density and maturation of APCs immediately after herpes simplex virus (HSV) inoculation. The purpose of this study was to investigate the role of corneal dendritic cells (DCs) and macrophages (MΦs) in acute HSV keratitis(HSK).

Methods: : Corneal DCs were locally depleted with subconjunctival (s.c.) injection of diphtheria toxin (DT, 30 ng) into CD11c-DTR-GFP C57BL/6 (B6) transgenic mice (DC(-)) 3 days before and every 3 days after inoculation. To deplete corneal MΦs, clodronate liposomes (10µl) were injected s.c. into B6 wild type (WT) mice (MΦ(-)). Results were compared to sham-treated WT control mice (n=6/group). After topical inoculation with HSV-1 McKrae strain, clinical opacity scores were graded daily. Corneas, trigeminal ganglia (TG) and draining lymph nodes (dLNs) were excised at days 1, 2, 3, 5 and 7 post-inoculation(p.i). Immunofluorescene staining of excised corneas was performed for β-tubulin and for HSV, and the nerve densities quantified. TG and dLNs were homogenized and specific HSV-1 gB mRNA were analyzed by qRT-PCR.

Results: : The corneal opacity scores in DC(-) (p<0.01), but not MΦ(-)(p>0.05) was more severe compared to WT. DC(-) demonstrated severe and rapid destruction of subbasal corneal nerves (44210±6560, 6820±1540 and 550±135µm/mm2 for days 1, 2 and 3 p.i respectively), as compared to WT (64200±8890,13980±3380 and 1340±520µm/mm2; p<0.05) and control mice (124500±46370µm/mm2), while MΦ(-) (61200±7960, 15730±3210 and 1130±430 µm/mm2; p>0.05) did not show statistical difference to WT. Viral transmission to TG was delayed in DC(-) (day 3) compared to WT and MΦ(-)(day 1). Compared to DC(-), relative HSV mRNA was increased 3.5-fold(p<0.05) in TG of WT and MΦ(-) at day 5 p.i, and increased 2-fold (p<0.05) in dLNs of WT and MΦ(-) compared to DC(-) at day 5 p.i.. Interestingly, HSV was also detected in the contralateral TG with 1 day delay, with similar decrease in HSV-1 mRNA levels in DC(-) mice.

Conclusions: : Corneal DCs, but not MΦ, decrease destruction of subbasal nerves and suppress HSV keratitis, while mediating systemic viral transmission.

Keywords: herpes simplex virus • cornea: basic science • keratitis 
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