April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
Olfactory Ensheathing Cells Promote Retinal Ganglion Cell Growth, Survival and Neurite Bundling
Author Affiliations & Notes
  • Sauparnika Vijay
    NIHR Biomedical Research Centre for Ophthalmology, Moorfields Eye Hospital and UCL Institute of Ophthalmology, London, United Kingdom
  • Annegret H. Dahlmann-Noor
    NIHR Biomedical Research Centre for Ophthalmology, Moorfields Eye Hospital and UCL Institute of Ophthalmology, London, United Kingdom
  • Richard Foxton
    UCL Institute of Ophthalmology, London, United Kingdom
  • Peter Munro
    UCL Institute of Ophthalmology, London, United Kingdom
  • Peng T. Khaw
    NIHR Biomedical Research Centre for Ophthalmology, Moorfields Eye Hospital and UCL Institute of Ophthalmology, London, United Kingdom
  • Footnotes
    Commercial Relationships  Sauparnika Vijay, None; Annegret H. Dahlmann-Noor, None; Richard Foxton, None; Peter Munro, None; Peng T. Khaw, None
  • Footnotes
    Support  Dorothy Hodgkins Postgraduate Award, Fight for Sight, Helen Hamlyn Trust, UK, the UK Medical Research Council, John Nolan, Freemasons Grand Charity, NIHR Biomedical Research Centre for Ophthalmology
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 3089. doi:
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      Sauparnika Vijay, Annegret H. Dahlmann-Noor, Richard Foxton, Peter Munro, Peng T. Khaw; Olfactory Ensheathing Cells Promote Retinal Ganglion Cell Growth, Survival and Neurite Bundling. Invest. Ophthalmol. Vis. Sci. 2011;52(14):3089.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract
 
Purpose:
 

Olfactory ensheathing cells (OEC) are glia residing in the olfactory mucosa and bulb, where they allow continuous regeneration of axons of olfactory receptor neurons. OEC support regeneration of axons in spinal injury models with functional regain. OEC promote regeneration of retinal ganglion cell (RGC) axons in-vitro; however, myelination or synapse formation have not been described. The aim of our study was to examine interactions between OEC and RGC in-vitro.

 
Methods:
 

We isolated OEC and RGC from postnatal rats. We selected primary RGC from a retinal cell suspension by magnetic sorting and cultured cells on glass coverslips in neurobasal medium enriched with neurotrophins. We maintained cells separately for five days. We characterized OEC and RGC by immunocytochemistry (ICC) and RT-PCR for specific markers. We added OEC to RGC cultures and co-cultured both for 14 days. We fixed the co-cultures and carried out ICC and transmission electron microscopy (TEM). We studied a subset of co-cultures with time-lapse phase microscopy.

 
Results:
 

We identified OEC on ICC by expression of p75 neurotrophin receptor, and RGC by neurofilament (NF). On TEM, OEC have large amounts of glycogen deposits; neurites have evenly spaced neurofilaments. RGC survival is enhanced by 204% in the presence of OEC, and neurites express more presynaptic puncta. TEM images show that OEC processes engulf synapses (figure). In the presence of RGCs OEC express synatotagmin protein, but not in monoculture. OEC organize RGC neurites into bundles with parallel fibres.

 
Conclusions:
 

OECs support RGC regeneration and may facilitate formation of functional synapses in-vitro. We have demonstrated for the first time that RGC synapses are enhanced by the physical presence of OEC processes, and that OEC organize RGC neurites into parallel bundles. Induction of RGC synpaptotagmin may indicate that OEC can modulate neuronal synaptic activity.  

 
Keywords: ganglion cells • neuroprotection • synapse 
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