April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
In Vivo Confocal Microscopy Study of Dendritic Cell Counts in Corneal Limbal Stem Cell Deficiency
Author Affiliations & Notes
  • Kristina Kurbanyan
    Cornea Division, Jules Stein Eye Institute, David Geffen School of Medicine at UCLA, Los Angeles, California
  • Sophie Deng
    Cornea Division, Jules Stein Eye Institute, David Geffen School of Medicine at UCLA, Los Angeles, California
  • Footnotes
    Commercial Relationships  Kristina Kurbanyan, None; Sophie Deng, None
  • Footnotes
    Support  Prevent Blindness of America
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 5117. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Kristina Kurbanyan, Sophie Deng; In Vivo Confocal Microscopy Study of Dendritic Cell Counts in Corneal Limbal Stem Cell Deficiency. Invest. Ophthalmol. Vis. Sci. 2011;52(14):5117.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : To quantify dendritic cells (DC) in patients with limbal stem cell deficiency (LSCD), utilizing in vivo confocal microscopy (IVCM).

Methods: : Retrospective analysis of IVCM (Heidelberg Retina Tomograph 3 with the Rostock Cornea Module [HRT3/RCM]) images of corneas with the diagnosis of limbal stem cell deficiency due to multiple surgeries (n=9), Stevens Johnson Syndrome (n=5), exposure keratopathy (n=3), neurotrophic keratopathy (n=3), chemical injury (n=2), dry eye (n=2), and medicamentosa (n=1). Dendritic cells in the superior, inferior, temporal and nasal limbus and central cornea were quantified and the average results were compared to 8 corneas of normal controls without history of contact lens wear. For each variable, results for three frames were averaged and analyzed utilizing the Student’s t-test.

Results: : The DC were mostly found within the subepithelial and the basal epithelial layers. The DC density in normal controls was 25.0 ± 2.2 /mm2 in the limbus and 14.3 ± 1.2 /mm2 centrally. In comparison to controls, the DC density among all groups (n=25) was found to be 8.9 ± 0.7 /mm2 (p=0.0001) in the limbus and 8.1 ± 0.7 /mm2 (p=0.0001) centrally. In the multiple surgery group, the DC density was 5.4 ± 0.4 /mm2 (p=0.0001) in the limbus and 16.6 ± 1.6 /mm2 (p=0.0047) centrally. In the Stevens Johnson Syndrome group, the DC density was 13.6 ± 1.3 /mm2 (p=0.0001) in the limbus and 4.7 ± 0.3 /mm2 (p=0.0001) centrally. In exposure keratopathy, the DC density was 4.0 ± 0.2 /mm2 (p=0.0001) in the limbus and 5.4 ± 0.3 /mm2 (p=0.0001) centrally. In chemical injury, the DC was 11.0 ± 1.0 /mm2 (p=0.0001) in the limbus and 23.3 ± 2.3 /mm2 (p=0.0001) centrally.

Conclusions: : DC density was overall lower in both the central cornea and limbus in LSCD when compared to normal controls, with the exception of iatrogenic and chemical injury induced LSCD in which the DC density was higher in the central cornea. The number of DC in the cornea and limbus alone may not be an important factor in LSCD pathogenesis.

Keywords: cornea: clinical science • microscopy: confocal/tunneling 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×