April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
Electroretinographic Evaluation of the Effect of Insulin Resistance Induced by Glucocorticoid Administration together with Inactivity and Overeating
Author Affiliations & Notes
  • Per Kappelgaard
    Department of Ophthalmology,
    Glostrup Hospital, University of Copenhagen, Copenhagen, Denmark
  • Katrine B. Hansen
    Department of Clinical Physiology,
    Glostrup Hospital, University of Copenhagen, Copenhagen, Denmark
  • Filip K. Knop
    Department of Internal Medicine F, Gentofte Hospital, Copenhagen, Denmark
  • Michael Larsen
    Department of Ophthalmology,
    Glostrup Hospital, University of Copenhagen, Copenhagen, Denmark
    Kennedy Center - National Eye Clinic, Glostrup, Copenhagen, Denmark
  • Footnotes
    Commercial Relationships  Per Kappelgaard, None; Katrine B. Hansen, None; Filip K. Knop, None; Michael Larsen, None
  • Footnotes
    Support  Danish Agency for Science, Technology, and Innovation (Grant no. 10-080573) and the Novo Nordisk Foundation
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 6081. doi:
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      Per Kappelgaard, Katrine B. Hansen, Filip K. Knop, Michael Larsen; Electroretinographic Evaluation of the Effect of Insulin Resistance Induced by Glucocorticoid Administration together with Inactivity and Overeating. Invest. Ophthalmol. Vis. Sci. 2011;52(14):6081.

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Abstract

Purpose: : To study normo- and hyperglycemic electrophysiological retinal function in healthy subjects before and after induction of temporary insulin resistance.

Methods: : Dark-adapted full field electroretinography (ffERG) was made in 10 eyes in 10 healthy men (age 25±3 years (mean±SD); body mass index 22±3 kg/m2; fasting plasma glucose 4.9±0.2 mM), while fasting and 2-3 hours later while plasma glucose was clamped at 10 mM. A baseline session in the subjects’ habitual state was followed by a second session after being on a high-caloric diet with minimal physical activity while taking prednisolone 37.5 mg/day for 9.1±1.4 days.

Results: : The intervention had no impact on fasting plasma glycemia, but the postprandial increase in glycemia 2 h into an oral glucose tolerance test rose from 4.2±0.8 to 5.5±1.5 mM (P=0.023) and the area under curve increased from 154±81 to 250±40 mM·120 min (P=0.021) concomitantly with increases in insulin resistance according to the homeostatic model assessment (P=0.011) and fasting plasma insulin levels (P=0.003). The amplitudes and implicit times of the ffERG did not vary significantly (P<0.05) in any components between normo- and hyperglycemia, nor did they vary with insulin resistance. Thus, the fasting b-wave amplitude of the combined rod-cone response was 432±84 mV at baseline and 463±71 mV (P=0.13) during increased insulin resistance while the hyperglycemic b-wave amplitude was 449±91 mV at baseline and 478±119 mV (P=0.55) during increased insulin resistance. Also, differences in b-wave amplitude between normo- and hyperglycemia at the baseline visit (P=0.62) and during insulin resistance (P=0.62) were non-significant.

Conclusions: : Neither hyperglycemia, nor short-term prednisolone-induced insulin resistance had any measurable effect on the components of the ffERG in healthy subjects.

Clinical Trial: : http://www.clinicaltrials.gov NCT01140932

Keywords: retina • electroretinography: clinical • drug toxicity/drug effects 
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