April 2011
Volume 52, Issue 14
Free
ARVO Annual Meeting Abstract  |   April 2011
9-cis Retinoic Acid Modulates Corneal Lymphangiogenesis
Author Affiliations & Notes
  • Tatiana Ecoiffier
    Center for Eye Disease and Development, Program in Vision Science, and School of Optometry, University of California Berkeley, Berkeley, California
  • Inho Choi
    Departments of Surgery and Biochemistry and Molecular Biology, Norris Comprehensive Cancer Center, University of Southern California, Los Angeles, California
  • Young Hong
    Departments of Surgery and Biochemistry and Molecular Biology, Norris Comprehensive Cancer Center, University of Southern California, Los Angeles, California
  • Sunju Lee
    Departments of Surgery and Biochemistry and Molecular Biology, Norris Comprehensive Cancer Center, University of Southern California, Los Angeles, California
  • Lu Chen
    Center for Eye Disease and Development, Program in Vision Science, and School of Optometry, University of California Berkeley, Berkeley, California
  • Footnotes
    Commercial Relationships  Tatiana Ecoiffier, None; Inho Choi, None; Young Hong, None; Sunju Lee, None; Lu Chen, None
  • Footnotes
    Support  This work is supported in part by research grants from NIH, DoD, and University of California at Berkeley (LC). T. Ecoiffier is an Ezell Fellow from the American Optometric Foundation.
Investigative Ophthalmology & Visual Science April 2011, Vol.52, 6388. doi:
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    • Get Citation

      Tatiana Ecoiffier, Inho Choi, Young Hong, Sunju Lee, Lu Chen; 9-cis Retinoic Acid Modulates Corneal Lymphangiogenesis. Invest. Ophthalmol. Vis. Sci. 2011;52(14):6388.

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Abstract

Purpose: : 9-cis retinoic acid (RA) is a metabolite of vitamin A and controls many biological and pathological processes. The purpose of this study is to use the unique tissue of the cornea to investigate potential roles of 9-cis RA in hemangiogenesis (HG; the development of new blood vessels), lymphangiogenesis (LG; the development of new lymphatic vessels), or both, which remain largely unknown to this stage.

Methods: : Uniform slow-release 9-cis RA or control pellets were implanted in normal adult BALB/c mouse corneas. Flat-mount corneas were harvested at Day 14 after the surgeries for immunofluorescent microscopic studies using specific antibodies against CD31 (a panendothelial cell marker) and LYVE-1 (a lymphatic marker). Digital images were analyzed by NIH Image J software. In addition, corneal HG was also evaluated by ophthalmic slit-lamp examinations.

Results: : As evaluated by the percentage of covered area, 9-cis RA triggered a significant in-growth of lymphatic vessels into the cornea (P = 0.005, n = 10). However, no statistical significance was detected in blood vessel development.

Conclusions: : This study provides the first evidence showing that 9-cis RA promotes LG in the cornea. Our data also demonstrate that in response to a certain pathological stimulation, corneal LG and HG can behave differently or independently. Further studies on this molecule may provide new mechanisms underlying differential processes of LG and HG and the development of novel strategies to treat lymphatic-related diseases, such as transplant rejection, cancer metastasis, inflammation, and lymphedema.

Keywords: neovascularization • cornea: basic science • transplantation 
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