April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Overexpression of Alpha A and Alpha B Crystallins Promote the Survival of Retinal Ganglion Cells After Optic Nerve Transection
Author Affiliations & Notes
  • N. Piri
    Ophthalmology, Jules Stein Eye Institute UCLA, Los Angeles, California
  • Y. Munemasa
    Ophthalmology, Jules Stein Eye Institute UCLA, Los Angeles, California
  • J. M. K. Kwong
    Ophthalmology, Jules Stein Eye Institute UCLA, Los Angeles, California
  • J. Caprioli
    Ophthalmology, Jules Stein Eye Institute UCLA, Los Angeles, California
  • Footnotes
    Commercial Relationships  N. Piri, None; Y. Munemasa, None; J.M.K. Kwong, None; J. Caprioli, None.
  • Footnotes
    Support  Gerald Oppenheimer Family Foundation (NP, JMKK), Nagai Foundation (YM), RPB (JC).
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 121. doi:
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      N. Piri, Y. Munemasa, J. M. K. Kwong, J. Caprioli; Overexpression of Alpha A and Alpha B Crystallins Promote the Survival of Retinal Ganglion Cells After Optic Nerve Transection. Invest. Ophthalmol. Vis. Sci. 2009;50(13):121.

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Abstract

Purpose: : Stress-induced crystallin expression is commonly viewed as an activation of the cell survival mechanism. We analyzed expression of alpha A and alpha B crystallins in a rat optic nerve transection (ONT) model characterized by specific retinal ganglion cell (RGC) degeneration and determined their role in RGC survival.

Methods: : ONT was performed on adult male Wistar rats weighing 300-350g. Quantitative and spatial expression were examined with western blot and immunohistochemisty, respectively. Electroporation was used to deliver alpha A and alpha B expression constructs to RGCs. Cell protective effects of alpha A and alpha B overexpression after ONT was determined with RGC density analysis.

Results: : Alpha A and alpha B mRNA and protein expression in the retina was observed predominantly in the ganglion cell layer, where the majority of crystallin-positive cells were co-localized with RGCs. Crystallin expression was also present in the inner nuclear and to a lesser degree in the outer nuclear layers. Very few crystallin-positive cells were found 14 days after ONT, which leads to more than 90% of RGCs loss. Quantitative analysis showed that the expression level of the alpha A (control vs ONT, n=8, P = 0.016) and alpha B proteins (control vs ONT, n=8, P = 0.005) was reduced by approximately 1.6 fold after ONT compared to the contralateral untreated retinas. The effect of alpha A and alpha B overexpression on RGC survival was evaluated 14 days after axotomy. The numbers of surviving RGCs after alpha A and alpha B transfection were 389±57 and 353.57±60 cells/mm2, respectively, versus 198±29 cells/mm2 following transfection with the vector alone [ONT + EGFP (n=5) vs ONT + alpha A - EGFP (n=8), P = 0.003; ONT + EGFP (n=5) vs ONT + alpha B - EGFP (n=8), P = 0.003] or 206±60 cells/mm2 in non-transfected retinas [ONT (n=9) vs ONT + alpha A - EGFP (n=8), P = 0.001; ONT (n=9) vs ONT + alpha B - EGFP (n=8), P = 0.001].

Conclusions: : The results indicate a significant neuroprotective effect of alpha-crystallins on RGCs with approximately 95% and 75% increase in the RGC survival rate mediated by alpha A and alpha B overexpression, respectively.

Keywords: ganglion cells • neuroprotection • crystallins 
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