April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
A High Content Screening Platform for the Measurement of Trophic Factors in Rodent Models of Retinitis Pigmentosa
Author Affiliations & Notes
  • E. Clerin
    Genetics, Institut de la vision, Paris, France
  • S. Mohand-Saïd
    Genetics, Institut de la vision, Paris, France
  • C. Jaillard
    Genetics, Institut de la vision, Paris, France
  • T. Cronin
    Genetics, Institut de la vision, Paris, France
  • J. Bennett
    Department of Ophtalmology, University of Pennsylvania, Philadelphia, Pennsylvania
  • J.-A. Sahel
    Genetics, Institut de la vision, Paris, France
  • T. Léveillard
    Genetics, Institut de la vision, Paris, France
  • Footnotes
    Commercial Relationships  E. Clerin, None; S. Mohand-Saïd, None; C. Jaillard, None; T. Cronin, None; J. Bennett, None; J.-A. Sahel, None; T. Léveillard, None.
  • Footnotes
    Support  Inserm, Fondation Fighting Blindness (FFB), ANR
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 1936. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      E. Clerin, S. Mohand-Saïd, C. Jaillard, T. Cronin, J. Bennett, J.-A. Sahel, T. Léveillard; A High Content Screening Platform for the Measurement of Trophic Factors in Rodent Models of Retinitis Pigmentosa. Invest. Ophthalmol. Vis. Sci. 2009;50(13):1936.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : We have developed an automated cell counting platform to validate the activity of the Rod-derived Cone Viability Factors, as therapeutic agents for the treatment of patients suffering from retinitis pigmentosa. The automated cone counting platform for retinal explants is used both for mouse and rat models of rod-cone degeneration. The specific objective was to validate this novel method by comparison to the semi-manual stereological counting used previously (Mohand-Saïd et al., 1998).

Methods: : Seven retinas from rd1 mouse (C3H) and their wild type isogenic controls (C3H+/+) were dissected from 15 to 90 days post-natal (PN), orientated and fixated with 4% paraformaldehyde. For each mouse, one eye was used for automated counting and the contralateral eye for the semi-manual stereological method. For both methods the cones were labelled with peanut agglutinin (PNA) coupled to Texas-Red. The novel method involves an inverted microscope linked to a computer driven motorized platform. Following automated acquisition in the depth of the tissue, cone density was measured using a special application of the Metamorph software.

Results: : The decrease in cone density following rod loss in the rd1 mouse was found to be equivalent using either the stereological method or the novel automated platform. The decrease in cone density from 35 to 90 days post-natal was also quantified in an additional rod-cone degenerative model, the rd10. Regionalisation in the cone density was demonstrated for the rd7 mouse model following labelling with polyclonal antibodies generated against short-wave opsin. We also used this platform to quantity the loss of cones mouse models carrying an inactivation of the genes encoding the Rod derived Cone Viability Factors.

Conclusions: : We demonstrate the usefulness of this platform for preclinical studies aimed at preventing the loss of central vision using neurotrophic factors.

Keywords: photoreceptors • gene screening • growth factors/growth factor receptors 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×