April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Formulation and Characterization of N-Acetyl Cysteine Loaded PLGA Nanoparticles for Neuroprotection to Retinal Ganglion Cells in an in vitro Model of Glaucomatous Cell Death
Author Affiliations & Notes
  • A. J. Ondricek
    Cell Biology and Genetics,
    University of North Texas Health Science Center, Fort Worth, Texas
  • A. Mukerjee
    Molecular Biology and Immunology,
    University of North Texas Health Science Center, Fort Worth, Texas
  • J. K. Vishwanatha
    Molecular Biology and Immunology,
    University of North Texas Health Science Center, Fort Worth, Texas
  • Footnotes
    Commercial Relationships  A.J. Ondricek, None; A. Mukerjee, None; J.K. Vishwanatha, None.
  • Footnotes
    Support  None.
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 2410. doi:
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      A. J. Ondricek, A. Mukerjee, J. K. Vishwanatha; Formulation and Characterization of N-Acetyl Cysteine Loaded PLGA Nanoparticles for Neuroprotection to Retinal Ganglion Cells in an in vitro Model of Glaucomatous Cell Death. Invest. Ophthalmol. Vis. Sci. 2009;50(13):2410.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : The eye is a preferential environment to study sustained delivery methods due to its isolation from peripheral circulation via the blood retina barrier. Likewise, poly-L-lactic-co glycolic acid (PLGA) is a preferential polymer to encapsulate drugs for sustained delivery because it is biodegradable, biocompatible and FDA approved. The purpose of this study is to formulate and characterize N-acetyl cysteine (NAC) loaded PLGA nanoparticles and study the effect of them delivering neuroprotective antioxidants to retinal ganglion cells. We hypothesize that NAC loaded PLGA nanoparticles (NAC-PLGA nps) will afford sustained neuroprotection to retinal ganglion cells (RGCs) via activation of cell survival pathways when compared to NAC alone.

Methods: : PLGA nanoparticles were synthesized utilizing double emulsion solvent evaporation technique. Nanoparticles were then characterized for size using a particle size analyzer, Nanotrac. Intracellular localization in RGC-5 and to the RGC cell layer in pig eyes was determined using Nile Red. Encapsulation efficiency was determined by direct method utilizing the thiol-reactive probe, monobromibimane. Cell survival assay was carried out using Calcien AM assay. Western blotting analysis was used for determination of cell signaling events.

Results: : NAC-PLGA nps were optimized based on preferred size (100-250 nm) and encapsulation efficiency. It was found that NAC-PLGA nps localize intracellularly in vitro in RGC-5s and ex vivo to RGCs in pig eyes. NAC-PLGA nps affords neuroprotection to RGC-5s against Iodoacetic Acid induced cytotoxicity. NAC-PLGA nps induce sustained phosphorylation of ERK1 and 2, and S6 over 24 hours compared with control.

Conclusions: : NAC-PLGA nanoparticles have potential to become an effective treatment, in vitro, against the generation of oxidative stress and cytotoxicity. They are a plausible for application in glaucoma because of the ability to localize intracellularly in RGCs. The application of such a sustained delivery method could improve patient outcomes and compliance when combined with IOP-lowering treatments or in normal tension glaucomas alone.

Keywords: antioxidants • neuroprotection 
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