April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Expression of Il-1alpha In Rabbit Lacrimal Gland After Corneal Injury
Author Affiliations & Notes
  • A. A. Esposito
    Dept Of Ophthalomology, Cole Eye Institute-Cleveland Clinic, Cleveland, Ohio
  • M. Salomao
    Dept Of Ophthalomology, Cole Eye Institute-Cleveland Clinic, Cleveland, Ohio
  • S. S. Chaurasia
    Dept Of Ophthalomology, Cole Eye Institute-Cleveland Clinic, Cleveland, Ohio
  • V. Agrawal
    Dept Of Ophthalomology, Cole Eye Institute-Cleveland Clinic, Cleveland, Ohio
  • S. E. Wilson
    Dept Of Ophthalomology, Cole Eye Institute-Cleveland Clinic, Cleveland, Ohio
  • Footnotes
    Commercial Relationships  A.A. Esposito, None; M. Salomao, None; S.S. Chaurasia, None; V. Agrawal, None; S.E. Wilson, None.
  • Footnotes
    Support  EY10056 and EY15638 from National Eye Institute, NIH, Bethesda, MD and Research to Prevent Blindness, New York, NY. Dr. Wilson, recipient of the Research to Prevent Blindness Physician-Scientist Award
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 4239. doi:
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    • Get Citation

      A. A. Esposito, M. Salomao, S. S. Chaurasia, V. Agrawal, S. E. Wilson; Expression of Il-1alpha In Rabbit Lacrimal Gland After Corneal Injury. Invest. Ophthalmol. Vis. Sci. 2009;50(13):4239.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To investigate lacrimal gland production of the pro-inflammatory modulator interleukin-1 alpha (IL-1 alpha).

Methods: : This study was performed in New Zealand white rabbits. Main lacrimal glands were collected from unwounded control eyes and from eyes at 24 hours after 7.0 mm corneal epithelial debridment wounds generated in conjunction with corneal riboflavin-UV treatments. The lacrimal glands were harvested, placed in OCT, and frozen. Sections were evaluated for IL-1 alpha using immunocytochemistry and control sections were incubated with 10X concentration IL-1 alpha blocking peptide.

Results: : Basal Il-1 alpha expression was observed in the lacrimal glands of eyes with uninjured corneas. IL-1 alpha levels increased in the main lacrimal gland after corneal injury.

Conclusions: : The lacrimal gland may provide another source for IL-1 alpha in tears. IL-1 alpha from the lacrimal gland may contribute to the many cellular effects mediated by IL-1 during the corneal response to injury.

Keywords: lacrimal gland • inflammation • immunohistochemistry 
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