April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Low Dose Periocular Insulin Treats Retinal Neurodegeneration in Experimental Diabetic Retinopathy
Author Affiliations & Notes
  • R. S. Singh
    Ophthalmology, Penn State Hershey Eye Center, Hershey, Pennsylvania
  • P. E. Fort
    Ophthalmology, Penn State Hershey Eye Center, Hershey, Pennsylvania
  • M. K. Losiewicz
    Ophthalmology, Penn State Hershey Eye Center, Hershey, Pennsylvania
  • T. W. Gardner
    Ophthalmology, Penn State Hershey Eye Center, Hershey, Pennsylvania
  • Footnotes
    Commercial Relationships  R.S. Singh, None; P.E. Fort, None; M.K. Losiewicz, None; T.W. Gardner, None.
  • Footnotes
    Support  JDRF, PA Lions Sight Conservation
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 5361. doi:
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    • Get Citation

      R. S. Singh, P. E. Fort, M. K. Losiewicz, T. W. Gardner; Low Dose Periocular Insulin Treats Retinal Neurodegeneration in Experimental Diabetic Retinopathy. Invest. Ophthalmol. Vis. Sci. 2009;50(13):5361.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To test the hypothesis that low dose periocular delivery of insulin to the retina of diabetic rats, may be neuroprotective in early diabetic retinopathy.

Methods: : Regular human insulin in a dose of 0.0325IU/100g body weight or vehicle (1xPBS) was injected subconjunctivally for 4 consecutive days in control and 1 month diabetic male Sprague Dawley rats. The biological effects of subconjunctival insulin on retinal neurodegeneration were analyzed by evaluating pro-survival Akt-1 signaling. Retinal cell death was assessed by TUNEL staining of flat mounted whole retinas.

Results: : Low dose subconjunctival insulin injections for 4 consecutive days did not have any effect on systemic blood glucose levels or animal weights. However, it restored pro-survival Akt-1 kinase activity (p<0.04) in the retina and reduced the rate of apoptosis (p=0.0004) seen in early diabetic retinopathy.

Keywords: diabetic retinopathy • neuroprotection • apoptosis/cell death 
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