April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Bruch's Membrane Proteome Alterations in Insulin-Dependent Diabetes Mellitus
Author Affiliations & Notes
  • S. Schaal
    Ophthalmology & Visual Sciences,
    University of Louisville, Louisville, Kentucky
  • J. Cai
    Pharmacology and Toxicology,
    University of Louisville, Louisville, Kentucky
  • L. Geng
    Ophthalmology & Visual Sciences,
    University of Louisville, Louisville, Kentucky
  • H. J. Kaplan
    Ophthalmology & Visual Sciences,
    University of Louisville, Louisville, Kentucky
  • T. H. Tezel
    Ophthalmology & Visual Sciences,
    University of Louisville, Louisville, Kentucky
  • Footnotes
    Commercial Relationships  S. Schaal, None; J. Cai, None; L. Geng, None; H.J. Kaplan, None; T.H. Tezel, None.
  • Footnotes
    Support  Supported (THT) in part by NIH (KO8EY0416120-01), a Career Development Award from Research to Prevent Blindness, Inc, NYC, NY and the KY Challenge Research Trust Fund (HJK).
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 5368. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      S. Schaal, J. Cai, L. Geng, H. J. Kaplan, T. H. Tezel; Bruch's Membrane Proteome Alterations in Insulin-Dependent Diabetes Mellitus. Invest. Ophthalmol. Vis. Sci. 2009;50(13):5368.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : To determine human Bruch’s membrane proteome changes specific to insulin-dependent diabetes mellitus (IDDM).

Methods: : Human choroid-Bruch’s membrane explants were prepared from young (<55 years-old) and fresh (<24 hours) donor eyes with and without IDDM. Cells were removed with 0.02 N ammonium hydroxide treatment leaving only the extracellular matrices. An excimer laser was then used to trim the choroid leaving only pure human Bruch’s membrane. 6.5 mm circular explants of Bruch’s membrane were further processed for protein extraction and separation using 2-D gel electrophoresis. Matrix-assisted laser desorption/ionization time-of-flight mass spectrometry (MALDI-TOF-MS) and electrospray ionization-quadruple time-of-flight MS/MS (ESI-Q-TOF MS/MS) were used to identify the mapped proteins. Quantitative and qualitative comparison of spots common in >2/3 of the gels were compared between donors with and without IDDM to determine the diabetes-specific differences in the Bruch’s membrane proteome. Results were confirmed with Western blotting analysis.

Results: : 261 proteins were identified in normal young Bruch’s membrane. 132 of these proteins did not reveal any qualitative or quantitative change among normal donors. IDDM was characterized with quantitative alteration of 78 (59.1%) of the common proteins. Among these proteins 53 (40.2%) were over-expressed and 25 (18.9%) were downregulated.

Conclusions: : IDDM induces specific changes in protein expression in human Bruch’s membrane. It is quite possible that these changes contribute to the cellular pathophysiologic abnormalities in diabetic retinopathy since neighboring cells depend on cell-matrix interactions for survival, proliferation and regulation of their function.

Keywords: Bruch's membrane • diabetes • proteomics 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×