April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Depletion of Lyve-1(+) Cells in the Conjunctiva by Clodronate-liposomes
Author Affiliations & Notes
  • M. I. Melhorn
    Ophthalmology - Angiogenesis Laboratory, Massachusetts Eye & Ear Infirmary and Harvard Medical School, Boston, Massachusetts
    Institute of Molecular Biology and Bioinformatics, Charité, Campus Benjamin Franklin, Freie Universität Berlin, Germany
  • S. Nakao
    Ophthalmology - Angiogenesis Laboratory, Massachusetts Eye & Ear Infirmary and Harvard Medical School, Boston, Massachusetts
  • A. Hafezi-Moghadam
    Ophthalmology - Angiogenesis Laboratory, Massachusetts Eye & Ear Infirmary and Harvard Medical School, Boston, Massachusetts
  • Footnotes
    Commercial Relationships  M.I. Melhorn, None; S. Nakao, None; A. Hafezi-Moghadam, None.
  • Footnotes
    Support  NIH grants HL086933 and AI050775, Massachusetts Lions Eye Research Fund Inc., Marion W. and Edward F. Knight AMD Fund, and Research to Prevent Blindness.
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 5519. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      M. I. Melhorn, S. Nakao, A. Hafezi-Moghadam; Depletion of Lyve-1(+) Cells in the Conjunctiva by Clodronate-liposomes. Invest. Ophthalmol. Vis. Sci. 2009;50(13):5519.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : The role of lymphatics in the eye is not well understood. LYVE-1 is a lymphatic marker and is also expressed on certain macrophage subpopulations in the normal eye. Clodronate-liposome (Cl2MDP-LIP) injection is an established method for macrophage depletion. However, whether this method depletes also LYVE-1+cells in conjunctiva is unknown. Here we investigate the effect of Cl2MDP-LIP injection into the subconjunctival space on LYVE-1+and CD11b+ cell numbers.

Methods: : We generated Cl2MDP-LIP or PBS-liposomes (PBS-LIP) and injected 1-1.5 µL of the liposome stock solution subconjunctivally into mouse eyes. 48 h after injection, eyes were harvested and fixed for 10 min in 4% PFA. Corneal flatmounts were prepared and immunohistochemistry stainings for LYVE-1 and CD11b were performed. Micrographs were obtained and analyzed using ImageJ software.

Results: : Injection of Cl2MDP-LIP or PBS-LIP into conjunctiva caused a significant reduction in LYVE-1+ cells outside of the vessels compared to untreated control (5.5±2.6, 14±2.1, 120±36; P=0.004 or P=0.006; n=6). Cl2MDP-LIP significantly reduced LYVE-1+ cell numbers, compared to PBS-LIP (P= 0.03).In limbal area and cornea, PBS-LIP or Cl2MDP-LIP caused a significant increase in CD11b+ cells compared to untreated control (540±65, 571±106, 161±24, P=0.02, n=6).In conjunctiva a significant increase of CD11b+ cells was observed for Cl2MDP-LIP and PBS-LIP, compared to untreated control (P=0.05 for Cl2MBP-LIP and P=0.01 for PBS-LIP; cell numbers: 172±61, 267±66, 10±5.3; n=6), while Cl2MDP-LIP compared to PBS-LIP were not significant (P=0.3; n=6).

Keywords: cornea: endothelium • inflammation • immunohistochemistry 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×