April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
BG2: Analysis of a T Cell Receptor Transgenic B6 Mouse Specific for B-Galactosidase
Author Affiliations & Notes
  • S. W. McPherson
    Department of Ophthalmology, University of Minnesota, Minneapolis, Minnesota
  • N. D. Heuss
    Department of Ophthalmology, University of Minnesota, Minneapolis, Minnesota
  • C. Norbury
    Department of Microbiology and Immunology, Penn State College of Medicine, Hershey, Pennsylvania
  • M. Theoret
    Surgery Branch, National Cancer Institute, Bethesda, Maryland
  • D. Palmer
    Surgery Branch, National Cancer Institute, Bethesda, Maryland
  • N. Restifo
    Surgery Branch, National Cancer Institute, Bethesda, Maryland
  • D. S. Gregerson
    Department of Ophthalmology, University of Minnesota, Minneapolis, Minnesota
  • Footnotes
    Commercial Relationships  S.W. McPherson, None; N.D. Heuss, None; C. Norbury, None; M. Theoret, None; D. Palmer, None; N. Restifo, None; D.S. Gregerson, None.
  • Footnotes
    Support  NIH Grants EY011374, EY011542, EY016376; Research to Prevent Blindness; and the Minnesota Lions and Lionesses Club
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 5924. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      S. W. McPherson, N. D. Heuss, C. Norbury, M. Theoret, D. Palmer, N. Restifo, D. S. Gregerson; BG2: Analysis of a T Cell Receptor Transgenic B6 Mouse Specific for B-Galactosidase. Invest. Ophthalmol. Vis. Sci. 2009;50(13):5924.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose: : Previously we developed and characterized a T cell receptor (TCR) transgenic (Tg) B10.A mouse (bgalTCR) specific for b-galactosidase (bgal) to study the immune response to bgal when expressed as a retinal neo-self antigen (Ag). Using these mice we observed mechanisms of immune regulation induced by retinal Ags that were different than those reported using the 3A9 TCR Tg T cell/retinal hen egg lysozyme Ag system (McPherson, et al., J. Immunol. 182; Ham, et al., IOVS. 45; and Lambe, et al., J. Immunol. 178). In order to confirm the validity of our model we examined the immune response to retinal bgal using a different TCR Tg mouse. Here we report on a B6 TCR-Tg mouse specific for bgal; BG2.

Methods: : A TCR Tg mouse on the B6 MHC background specific for a class II MHC restricted (CD4+ T cell) epitope of bgal was developed. Identification of the BG2 T cell epitope was done by in vitro proliferation and cytokine assays using overlapping peptides of bgal. Pathogenicity of the epitope was assayed in adaptive transfer of peptide-stimulated BG2 T cells. The regulatory T cell (Treg) immune regulation induced by retinal expression of bgal was analyzed by comparing the DTH (ear swelling) response of BG2 mice to B6arrbgal (Tg photoreceptor cell bgal expression) x BG2 double Tg mice.

Results: : Two peptides of bgal containing the common epitope of SVTLPAASHAI induced proliferation and production of IL-2, IFNg, and TNFa by BG2 T cells. Adoptive transfer of BG2 T cells stimulated with either peptide induced experimental autoimmune uveoretinitis (EAU) in B6arrbgal mice. B6arrbgal x BG2 double Tg mice exhibited reduced ear swelling compared to BG2 mice indicative of bgal-specific regulatory immune response associated with its expression in photoreceptor cells. All of these results are similar to those obtained using bgalTCR mice.

Conclusions: : We have developed and characterized the immune response of a B6 TCR Tg mouse specific for a retinal neo-self antigen. The fact that we obtained similar results with both our B10.A and B6 TCR Tg mice supports our findings that photoreceptor cell Ags induce a Treg mediated immune regulation.

Keywords: immunomodulation/immunoregulation • immune tolerance/privilege • autoimmune disease 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×