April 2009
Volume 50, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2009
Phenotype and Genotype in Danish Families With Best Vitelliform Macular Dystrophy
Author Affiliations & Notes
  • P. S. Schatz
    Ophthalmology, Lund University Hospital, Lund, Sweden
    Ophthalmology, Glostrup Hospital, University of Copenhagen, Denmark
  • S. K. Holfort
    Ophthalmology, Glostrup Hospital, University of Copenhagen, Denmark
  • B. Sander
    Ophthalmology, Glostrup Hospital, University of Copenhagen, Denmark
  • M. Hellstrom Pigg
    Clinical Genetics, Uppsala University Hospital, Uppsala, Sweden
  • M. Larsen
    Ophthalmology, Glostrup Hospital, University of Copenhagen, Denmark
  • Footnotes
    Commercial Relationships  P.S. Schatz, None; S.K. Holfort, None; B. Sander, None; M. Hellstrom Pigg, None; M. Larsen, None.
  • Footnotes
    Support  stiftelsen för synskadade i f d Malmöhus län
Investigative Ophthalmology & Visual Science April 2009, Vol.50, 6266. doi:
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      P. S. Schatz, S. K. Holfort, B. Sander, M. Hellstrom Pigg, M. Larsen; Phenotype and Genotype in Danish Families With Best Vitelliform Macular Dystrophy. Invest. Ophthalmol. Vis. Sci. 2009;50(13):6266.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To describe the genotype and phenotype in Danish patients with Best Vitelliform Macular Dystrophy (BMD).

Methods: : Standard clinical ophthalmic investigation was performed. Electrophysiological investigations included electro-oculography (EOG), full-field ERG (ffERG) and multifocal ERG (mfERG). Imaging included optical coherence tomography (OCT) and fundus autofluorescence photography. Molecular genetic investigations included sequencing of BEST1 gene exons 2,4,6 and 8.

Results: : Depending on stage, several different patterns of structural alterations were found in patients, ranging from thickening of the choroid-pigment epithelium-photoreceptor complex, through subretinal fluid, and retinal atrophy. FfERG was normal. All patients had reduced Arden ratios by EOG. MfERG showed reduction of central retinal function. Mutation findings included c.[275G>A] leading to p.[Arg92His], c.[936C>A] leading to p.[Asp312Glu], c.[C349G] leading to p.[Leu82Val], and c.[253T>C] leading to p.[Thr85His].

Conclusions: : Phenotype varied according to stage of disease. Novel mutations were found in BEST1.

Keywords: retinal degenerations: hereditary • electroretinography: clinical • imaging methods (CT, FA, ICG, MRI, OCT, RTA, SLO, ultrasound) 
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