April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Relationship Between Clinical Course of DSAEK and Pathological Findings of Removed Endothelial Graft
Author Affiliations & Notes
  • H. Eguchi
    Ophthalmology, Institute of Health Biosciences, The University of Tokushima, Tokushima, Japan
  • K. Senba
    Ophthalmology, Institute of Health Biosciences, The University of Tokushima, Tokushima, Japan
  • T. Miyamoto
    Ophthalmology, Institute of Health Biosciences, The University of Tokushima, Tokushima, Japan
  • M. Matsubara
    Matsubara Eye Clinic, Ono-City, Japan
  • Footnotes
    Commercial Relationships  H. Eguchi, None; K. Senba, None; T. Miyamoto, None; M. Matsubara, None.
  • Footnotes
    Support  None.
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 778. doi:
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    • Get Citation

      H. Eguchi, K. Senba, T. Miyamoto, M. Matsubara; Relationship Between Clinical Course of DSAEK and Pathological Findings of Removed Endothelial Graft. Invest. Ophthalmol. Vis. Sci. 2010;51(13):778.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose: : To evaluate the relationship between postoperative clinical course of descemet’s stripping automated endothelial keratoplasty (DSAEK) and the pathological findings of removed endothelial graft.Patients and

Methods: : Three cases that underwent previous DSAEK and second surgery were recruited in this study. All cases had endothelial graft removal. Case 1 had a previous penetrating keratoplasty (PK) twenty years ago. He underwent a DSAEK for graft failure following long lived corneal graft. Although endothelial graft was thin and clear at 11 months postoperatively, PK was performed for the graft dislocation despite the 2 corrections. Case 2 had a previous PK 10 years ago and underwent DSAEK for graft failure following long lived corneal graft. A correction with gas tamponade was performed a day postoperatively for graft detachment, and PK was performed for graft failure 2 years postoperatively without episode of high intraocular pressure and rejection. Case 3 had previous DSAEKs twice for bullous keratopathy after lensectomy and 2 vitrectomies for trauma. Stromal edema and opacity suggestive of rejection was occurred after steroid tapering a month postoperatively. All endothelial grafts were 10% formalin fixed just after removal, and the microsection were prepared for hematoxllin-eosin staining and TdT-mediated dUTP-biotin nick end labeling (TUNEL) assay a day after removal.

Results: : In case 1, large number of keratocytes and endothelial cells were found and no inflammatory cell was existed in the graft. In case 2, although keratocytes were found, no endothelial cell was existed. In case 3, few keratocyte was found and no endothelial cell was existed. Some inflammatory cells in the lower layer of stroma and fibrin like structure at descemet’s membrane were found. Although no TUNEL-positive cell was found in case 1, some TUNEL-positive keratocytes were found in the lower stroma in case 2 and 3.

Conclusions: : The clinical courses of three cases were associated with their pathological findings of removed endothelial grafts. Keratocyte apoptosis in the lower layer of stroma may be associated with endothelial graft failure.

Keywords: cornea: clinical science • apoptosis/cell death 
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