April 2010
Volume 51, Issue 13
Free
ARVO Annual Meeting Abstract  |   April 2010
Optic Nerve Head (ONH) Lamina Cribrosa Insertion Migration and Pialization in Early Non-Human Primate (NHP) Experimental Glaucoma
Author Affiliations & Notes
  • H. Yang
    Devers Eye Institute, Portland, Oregon
  • G. Williams
    Devers Eye Institute, Portland, Oregon
  • J. Downs
    Devers Eye Institute, Portland, Oregon
  • I. Sigal
    Devers Eye Institute, Portland, Oregon
  • M. Roberts
    Devers Eye Institute, Portland, Oregon
  • J. Grimm
    Devers Eye Institute, Portland, Oregon
  • H. Thompson
    School of Public Health, LSUHSC, New Orleans, Louisiana
  • C. Burgoyne
    Devers Eye Institute, Portland, Oregon
  • Footnotes
    Commercial Relationships  H. Yang, None; G. Williams, None; J. Downs, None; I. Sigal, None; M. Roberts, None; J. Grimm, None; H. Thompson, None; C. Burgoyne, None.
  • Footnotes
    Support  EY 011610
Investigative Ophthalmology & Visual Science April 2010, Vol.51, 1631. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      H. Yang, G. Williams, J. Downs, I. Sigal, M. Roberts, J. Grimm, H. Thompson, C. Burgoyne; Optic Nerve Head (ONH) Lamina Cribrosa Insertion Migration and Pialization in Early Non-Human Primate (NHP) Experimental Glaucoma. Invest. Ophthalmol. Vis. Sci. 2010;51(13):1631.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract
 
Purpose:
 

To geometrically characterize the lamina cribrosa insertion into the scleral canal wall in normal and early experimental glaucoma (EEG) NHP ONH to test whether regional posterior (outward) migration of the lamina cribrosa insertion occurs in NHP EEG leading to pialization (laminar beam insertion into the pia instead of the sclera).

 
Methods:
 

Trephinated ONHs from both eyes of 12 normal (N) and 9 unilateral EEG NHPs were perfusion fixed, serial sectioned, 3D reconstructed and delineated. Regional and overall values for the following distances were calculated for each ONH: anterior scleral canal opening to anterior laminar insertion distance(ASCO-ALI); anterior to posterior laminar insertion distance (ALI-PLI); and posterior scleral canal opening to posterior laminar insertion distance (PSCO-PLI). The outward ALI and PLI migrations were defined to be statistically significant changes in ASCO-ALI and PSCO-PLI distances in the outward direction. Data were pooled into 4 groups based on perfusion IOP and eye status: N10/10 (n=6); N 10/30or45 (n=6); EEG 10/10 (n=3) and EEG 10/30or45 (n=6). Treatment and region effects were accessed within each NHP and overall by ANOVA.

 
Results:
 

ALI was outwardly migrated in the EEG 30/45 group (P<0.0001). PLI was outwardly migrated in EEG 10/10 (22 µm) and EEG 30/45 (44 µm) (P<0.0001) groups. Laminar insertion thickening (ALI-PLI expansion) was significant (32 µm in both EEG groups). These changes of EEG eyes held true in a majority of EEG monkeys. Regionally, 3 of the 9 EEG eyes demonstrated outward PLI migration to the degree of posterior lamina pialization (Fig 1) in at least 2 adjacent regions.

 
Conclusions:
 

Outward migration of the ALI may be the result of physical trauma and/or remodeling of the anterior laminar beams. Outward migration of the PLI may be the result of laminar remodeling that includes retrolaminar septal recruitment (Roberts et al, IOVS, 2009). The implications of these findings on the pathogenesis of ONH splinter hemorrhages, cupping, blood flow alterations and axon loss will be discussed.  

 
Keywords: optic nerve • lamina cribrosa • sclera 
×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×